Amanote Research
Register
Sign In
Invasion and Metastasis of Oral Cancer Cells Require Methylation of E-Cadherin and/or Degradation of Membranous -Catenin
Clinical Cancer Research
- United States
doi 10.1158/1078-0432.ccr-04-0372
Full Text
Open PDF
Abstract
Available in
full text
Categories
Cancer Research
Oncology
Date
August 15, 2004
Authors
Y. Kudo
Publisher
American Association for Cancer Research (AACR)
Related search
Selective Degradation of E-Cadherin and Dissolution of E-Cadherin-Catenin Complexes in Epithelial Ischemia
American Journal of Physiology - Renal Physiology
Urology
Physiology
Reduction in Membranous Expression of Β-Catenin and Increased Cytoplasmic E-Cadherin Expression Predict Poor Survival in Gastric Cancer
British Journal of Cancer
Cancer Research
Oncology
Β-Catenin Mediates Alteration in Cell Proliferation, Motility and Invasion of Prostate Cancer Cells by Differential Expression of E-Cadherin and Protein Kinase D1
Journal of Cellular Biochemistry
Biochemistry
Cell Biology
Molecular Biology
Octamer-Binding Protein 4 Affects the Cell Biology and Phenotypic Transition of Lung Cancer Cells Involving Β-Catenin/E-Cadherin Complex Degradation
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
Concomitant Loss of P120-Catenin and Β-Catenin Membrane Expression and Oral Carcinoma Progression With E-Cadherin Reduction
PLoS ONE
Multidisciplinary
Ubiquitin-Like Protein FAT10 Promotes the Invasion and Metastasis of Hepatocellular Carcinoma by Modifying -Catenin Degradation
Cancer Research
Cancer Research
Oncology
Acquired Expression of NFATc1 Downregulates E-Cadherin and Promotes Cancer Cell Invasion
Cancer Research
Cancer Research
Oncology
Promoter Methylation of P16, RARβ, E-Cadherin, Cyclin A1 and Cytoglobin in Oral Cancer: Quantitative Evaluation Using Pyrosequencing
British Journal of Cancer
Cancer Research
Oncology
Competitive Regulation of E-Cadherin JuxtaMembrane Domain Degradation by P120-Catenin Binding and Hakai-Mediated Ubiquitination
PLoS ONE
Multidisciplinary
