Optimal Tumor Necrosis Factor Induction by Plasmodium Falciparum Requires the Highly Localized Release of Parasite Products

Infection and Immunity - United States
doi 10.1128/iai.71.6.3155-3164.2003
Full Text
Abstract

Available in full text

Date
Authors
Publisher

American Society for Microbiology

Related search

Invasion of Mouse Erythrocytes by the Human Malaria Parasite, Plasmodium Falciparum

Journal of Experimental Medicine
MedicineAllergyImmunology
1987English

The Role of Complement, Antibody, and Tumor Necrosis Factor Alpha in the Killing of Plasmodium Falciparum by the Monocytic Cell Line THP-1.

Infection and Immunity
ParasitologyInfectious DiseasesImmunologyMicrobiology
1997English

Cutting Edge: FHR-1 Binding Impairs Factor H–Mediated Complement Evasion by the Malaria Parasite Plasmodium Falciparum

Journal of Immunology
AllergyImmunology
2018English

The Malaria Parasite Plasmodium Vivax Exhibits Greater Genetic Diversity Than Plasmodium Falciparum

Nature Genetics
Genetics
2012English

Induction of Plasmodium Falciparum Strain 2300 Dormant Forms by Artemisinin

Universa Medicina
2016English

Inhibition of Human Erythroid Colony-Forming Units by Tumor Necrosis Factor Requires Beta Interferon.

Journal of Clinical Investigation
Medicine
1993English

Induction of Endogenous Tumor Necrosis Factor in Hepatocellular Carcinoma.

Acta Hepatologica Japonica
Hepatology
1989English

CORRIGENDUM: Genome Variation and Evolution of the Malaria Parasite Plasmodium Falciparum

Nature Genetics
Genetics
2007English

Cytokine Induction by Immunomodulatory Epitopes in S-Antigen and Tumor Necrosis Factor Alpha

Current Eye Research
Molecular NeuroscienceOphthalmologySensory SystemsCellular
1992English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy