(PDF) ATR Kinase Inhibition Leads to Durable

ATR Kinase Inhibition Leads to Durable Radiosensitization of the Murine Kras G12D /Twist1 Lung Adenocarcinoma Model via a CD8 + T Cell-Dependent Mechanism

International Journal of Radiation Oncology Biology Physics - Netherlands

doi 10.1016/j.ijrobp.2017.06.084

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October 1, 2017

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Elsevier BV

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Amanote Research

ATR Kinase Inhibition Leads to Durable Radiosensitization of the Murine Kras G12D /Twist1 Lung Adenocarcinoma Model via a CD8 + T Cell-Dependent Mechanism

International Journal of Radiation Oncology Biology Physics - Netherlands

doi 10.1016/j.ijrobp.2017.06.084

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Effective Use of PI3K and MEK Inhibitors to Treat Mutant Kras G12D and PIK3CA H1047R Murine Lung Cancers

CD8+ Memory T-Cell Inflation Renders Compromised CD4+ T-Cell-Dependent CD8+ T-Cell Immunity via Naïve T-Cell Anergy

Notch1Gene Mutations Target KRAS G12d-Expressing CD8+Cells and Contribute to Their Leukemogenic Transformation

Dendritic Cell-Secreted Lipocalin2 Induces CD8+ T-Cell Apoptosis, Contributes to T-Cell Priming and Leads to a TH1 Phenotype

Precise Spatio-Temporal Interruption of Regulatory T Cell-Mediated CD8+ T Cell Suppression Leads to Tumor Immunity

A Murine Xenograft Model of Spontaneous Metastases of Human Lung Adenocarcinoma

CD4+CD25+ Regulatory T Cells Suppress Allograft Rejection Mediated by Memory CD8+ T Cells via a CD30-dependent Mechanism

Acanthamoeba Castellanii Induces Host Cell Death via a Phosphatidylinositol 3-Kinase-Dependent Mechanism

ΙκΒ Kinase Α Is Required for Development and Progression of KRAS-mutant Lung Adenocarcinoma.

Amanote Research

ATR Kinase Inhibition Leads to Durable Radiosensitization of the Murine Kras G12D /Twist1 Lung Adenocarcinoma Model via a CD8 + T Cell-Dependent Mechanism

International Journal of Radiation Oncology Biology Physics - Netherlands

doi 10.1016/j.ijrobp.2017.06.084

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Effective Use of PI3K and MEK Inhibitors to Treat Mutant Kras G12D and PIK3CA H1047R Murine Lung Cancers

CD8+ Memory T-Cell Inflation Renders Compromised CD4+ T-Cell-Dependent CD8+ T-Cell Immunity via Na&iuml;ve T-Cell Anergy

Notch1Gene Mutations Target KRAS G12d-Expressing CD8+Cells and Contribute to Their Leukemogenic Transformation

Dendritic Cell-Secreted Lipocalin2 Induces CD8+ T-Cell Apoptosis, Contributes to T-Cell Priming and Leads to a TH1 Phenotype

Precise Spatio-Temporal Interruption of Regulatory T Cell-Mediated CD8+ T Cell Suppression Leads to Tumor Immunity

A Murine Xenograft Model of Spontaneous Metastases of Human Lung Adenocarcinoma

CD4+CD25+ Regulatory T Cells Suppress Allograft Rejection Mediated by Memory CD8+ T Cells via a CD30-dependent Mechanism

Acanthamoeba Castellanii Induces Host Cell Death via a Phosphatidylinositol 3-Kinase-Dependent Mechanism

ΙκΒ Kinase Α Is Required for Development and Progression of KRAS-mutant Lung Adenocarcinoma.

CD8+ Memory T-Cell Inflation Renders Compromised CD4+ T-Cell-Dependent CD8+ T-Cell Immunity via Naïve T-Cell Anergy