Loss of Family With Sequence Similarity 13, Member a Exacerbates Pulmonary Hypertension Through Accelerating Endothelial-To-Mesenchymal Transition

doi 10.1101/850115
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Cold Spring Harbor Laboratory

Related search

Endothelial-Specific Loss of IFT88 Promotes Endothelial-To-Mesenchymal Transition and Exacerbates Bleomycin-Induced Pulmonary Fibrosis

Scientific Reports
Multidisciplinary
2020English

Endothelial-To-Mesenchymal Transition in Pulmonary Hypertension

Circulation
Cardiovascular MedicinePhysiologyCardiology
2015English

FAM107A (Family With Sequence Similarity 107, Member A)

Atlas of Genetics and Cytogenetics in Oncology and Haematology
Cancer ResearchOncologyGeneticsHematology
2012English

Family With Sequence Similarity 129, Member B (FAM129B; MINERVA)

Science-Business eXchange
2009English

Fam57b(Family With Sequence Similarity 57, Member B), a Novel Peroxisome Proliferator-Activated Receptor Γ Target Gene That Regulates Adipogenesis Through Ceramide Synthesis

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2012English

Transition of Mesenchymal Stem/Stromal Cells to Endothelial Cells

Stem Cell Research and Therapy
GeneticsCell BiologyMolecular BiologyBiochemistryMedicineMolecular Medicine
2013English

Accelerating Neuroimage Registration Through Parallel Computation of Similarity Metric

PLoS ONE
Multidisciplinary
2015English

Dyssynchronous Pacing Triggers Endothelial-Mesenchymal Transition Through Heterogeneity of Mechanical Stretch in a Canine Model

Circulation Journal
MedicineCardiovascular MedicineCardiology
2014English

Endocardial Fibroelastosis Is Caused by Aberrant Endothelial to Mesenchymal Transition

Circulation Research
Cardiovascular MedicinePhysiologyCardiology
2015English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy