Amanote Research
Register
Sign In
Sustained Akt Signaling in Articular Chondrocytes Causes Osteoarthritis via Oxidative Stress-Induced Senescence in Mice
Bone Research
- United Kingdom
doi 10.1038/s41413-019-0062-y
Full Text
Open PDF
Abstract
Available in
full text
Categories
Endocrinology
Physiology
Histology
Metabolism
Diabetes
Date
August 5, 2019
Authors
Jing Xie
Jingting Lin
Min Wei
Yan Teng
Qi He
Guan Yang
Xiao Yang
Publisher
Springer Science and Business Media LLC
Related search
Sustained Oxidative Stress Causes Late Acute Renal Failure via Duplex Regulation on P38 MAPK and Akt Phosphorylation in Severely Burned Rats
PLoS ONE
Multidisciplinary
Transcription Factor Nfat1 Deficiency Causes Osteoarthritis Through Dysfunction of Adult Articular Chondrocytes
Journal of Pathology
Forensic Medicine
Pathology
FGFR3 Signaling Induces a Reversible Senescence Phenotype in Chondrocytes Similar to Oncogene-Induced Premature Senescence
Bone
Endocrinology
Physiology
Histology
Metabolism
Diabetes
FNDC5 Alleviates Oxidative Stress and Cardiomyocyte Apoptosis in Doxorubicin-Induced Cardiotoxicity via Activating AKT
Cell Death and Differentiation
Cell Biology
Molecular Biology
Chandipura Virus Induced Neuronal Apoptosis via Calcium Signaling Mediated Oxidative Stress
Frontiers in Microbiology
Microbiology
Inhibition of Β‐catenin Signaling in Articular Chondrocytes Results in Articular Cartilage Destruction
Arthritis & Rheumatism
Lithium Chloride Dependent Glycogen Synthase Kinase 3 Inactivation Links Oxidative DNA Damage, Hypertrophy and Senescence in Human Articular Chondrocytes and Reproduces Chondrocyte Phenotype of Obese Osteoarthritis Patients
PLoS ONE
Multidisciplinary
Fucoxanthin Alleviates Oxidative Stress Through Akt/Sirt1/FoxO3α Signaling to Inhibit HG-Induced Renal Fibrosis in GMCs
Marine Drugs
Drug Discovery
Bacterial Infection Causes Stress-Induced Memory Dysfunction in Mice
Gut
Gastroenterology
