Neuronal Gap Junction Coupling as the Primary Determinant of the Extent of Glutamate-Mediated Excitotoxicity

Journal of Neural Transmission - Germany
doi 10.1007/s00702-013-1109-7
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

Cut-Loading: A Useful Tool for Examining the Extent of Gap Junction Tracer Coupling Between Retinal Neurons

Journal of Visualized Experiments
ImmunologyMolecular BiologyBiochemistryMicrobiology Chemical EngineeringNeuroscienceGenetics
2012English

Glutamate-Mediated Excitotoxicity in Schizophrenia: A Review

European Neuropsychopharmacology
PsychiatryMental HealthNeurologyBiological PsychiatryPharmacology
2014English

Coordination of Neuronal Activity in Developing Visual Cortex by Gap Junction-Mediated Biochemical Communication

Journal of Neuroscience
Neuroscience
1998English

Na-K-Cl Cotransporter Contributes to Glutamate-Mediated Excitotoxicity

Journal of Neuroscience
Neuroscience
2003English

Gap Junction Coupling and Calcium Waves in the Pancreatic Islet

Biophysical Journal
Biophysics
2008English

Screening of Gap Junction Antagonists on Dye Coupling in the Rabbit Retina

Visual Neuroscience
Sensory SystemsPhysiology
2007English

L-Lactate-Mediated Neuroprotection Against Glutamate-Induced Excitotoxicity Requires ARALAR/AGC1

Journal of Neuroscience
Neuroscience
2016English

Calcium-Dependent Binding of Calmodulin to Neuronal Gap Junction Proteins

Biochemical and Biophysical Research Communications
BiochemistryCell BiologyMolecular BiologyBiophysics
2005English

Ischemia-Triggered Glutamate Excitotoxicity From the Perspective of Glial Cells

Frontiers in Cellular Neuroscience
Molecular NeuroscienceCellular
2020English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy