(PDF) 55- And 75-Kilodalton Tumor Necrosis Factor Receptors

55- And 75-Kilodalton Tumor Necrosis Factor Receptors Mediate Distinct Actions in Regard to Human Immunodeficiency Virus Type 1 Replication in Primary Human Macrophages.

Journal of Virology - United States

doi 10.1128/jvi.71.5.4150-4156.1997

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Date

January 1, 1997

Authors

G Herbein

S Gordon

Publisher

American Society for Microbiology

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Amanote Research

55- And 75-Kilodalton Tumor Necrosis Factor Receptors Mediate Distinct Actions in Regard to Human Immunodeficiency Virus Type 1 Replication in Primary Human Macrophages.

Journal of Virology - United States

doi 10.1128/jvi.71.5.4150-4156.1997

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Nitric Oxide Synthesis Enhances Human Immunodeficiency Virus Replication in Primary Human Macrophages

Interleukin 13 Inhibits Human Immunodeficiency Virus Type 1 Production in Primary Blood-Derived Human Macrophages in Vitro

Sustained Small Interfering RNA-Mediated Human Immunodeficiency Virus Type 1 Inhibition in Primary Macrophages

Vif-Negative Human Immunodeficiency Virus Type 1 Persistently Replicates in Primary Macrophages, Producing Attenuated Progeny Virus.

Endothelial Cells Enhance Human Immunodeficiency Virus Type 1 Replication in Macrophages Through a C/EBP-Dependent Mechanism

A Functional Role for ADAM10 in Human Immunodeficiency Virus Type-1 Replication

The Relationship Between Tumor Necrosis Factor and Human Immunodeficiency Virus Gene Expression in Lymphoid Tissue.

Distinct Domains of IkappaB-alpha Inhibit Human Immunodeficiency Virus Type 1 Replication Through NF-kappaB and Rev.

Augmentation of Virus Secretion by the Human Immunodeficiency Virus Type 1 Vpu Protein Is Cell Type Independent and Occurs in Cultured Human Primary Macrophages and Lymphocytes.