Amanote Research
Register
Sign In
Multifactorial Pathologies Promote Inflammation and Enhance Vulnerability to Late-Onset Alzheimer’s Disease: Implications for Possible Therapeutic Targets
doi 10.2174/9781608058709114020006
Full Text
Open PDF
Abstract
Available in
full text
Date
February 7, 2014
Authors
Unknown
Publisher
BENTHAM SCIENCE PUBLISHERS
Related search
Sphingolipids in Inflammation: Pathological Implications and Potential Therapeutic Targets
British Journal of Pharmacology
Pharmacology
Relationship Between Adiponectin Gene Polymorphisms and Late-Onset Alzheimer’s Disease
PLoS ONE
Multidisciplinary
Association of the Neprilysin Gene With Susceptibility to Late-Onset Alzheimer’s Disease
Dementia and Geriatric Cognitive Disorders
Psychiatry
Gerontology
Geriatrics
Mental Health
Cognitive Neuroscience
Microglial Inflammation and Phagocytosis in Alzheimer's Disease: Potential Therapeutic Targets
British Journal of Pharmacology
Pharmacology
The Ubiquitin-Proteasome System: Potential Therapeutic Targets for Alzheimer’s Disease and Spinal Cord Injury
Frontiers in Molecular Neuroscience
Molecular Neuroscience
Molecular Biology
Cellular
Investigation of 15 of the Top Candidate Genes for Late-Onset Alzheimer’s Disease
Human Genetics
Genetics
Age-At-Onset Linkage Analysis in Caribbean Hispanics With Familial Late-Onset Alzheimer’s Disease
Neurogenetics
Molecular Neuroscience
Genetics
Cellular
The Interactions of P53 With Tau and Aß Represent Potential Therapeutic Targets for Alzheimer’s Disease
Progress in Neurobiology
Neuroscience
Disease-Associated Particulates and Joint Inflammation; Mechanistic Insights and Potential Therapeutic Targets
Frontiers in Immunology
Allergy
Immunology
