(PDF) Impaired STING Pathway in Human Osteosarcoma U2OS

Impaired STING Pathway in Human Osteosarcoma U2OS Cells Contributes to the Growth of ICP0-Null Mutant Herpes Simplex Virus

Journal of Virology - United States

doi 10.1128/jvi.00006-17

Full Text

Open PDF

Abstract

Available in full text

Date

February 8, 2017

Authors

Thibaut Deschamps

Maria Kalamvoki

Publisher

American Society for Microbiology

Related search

Complementation of a Herpes Simplex Virus ICP0 Null Mutant by Varicella-Zoster Virus ORF61p

English

Herpes Simplex Virus VP16, but Not ICP0, Is Required to Reduce Histone Occupancy and Enhance Histone Acetylation on Viral Genomes in U2OS Osteosarcoma Cells

English

An Activity Specified by the Osteosarcoma Line U2OS Can Substitute Functionally for ICP0, a Major Regulatory Protein of Herpes Simplex Virus Type 1.

English

The Replication Defect of ICP0-Null Mutant Herpes Simplex Virus 1 Can Be Largely Complemented by the Combined Activities of Human Cytomegalovirus Proteins IE1 and Pp71

English

Ubiquitin-Specific Protease 9X in Host Cells Interacts With Herpes Simplex Virus 1 ICP0

Journal of Veterinary Medical Science

Veterinary

2016

English

The Null Mutant of the U(L)31 Gene of Herpes Simplex Virus 1: Construction and Phenotype in Infected Cells.

English

Varicella-Zoster Virus Gene 51 Complements a Herpes Simplex Virus Type 1 UL9 Null Mutant.

English

Theabrownin Triggers DNA Damage to Suppress Human Osteosarcoma U2OS Cells by Activating P53 Signalling Pathway

Journal of Cellular and Molecular Medicine

Molecular Medicine

Cell Biology

2018

English

Overexpression of the Herpes Simplex Virus Type 1 Immediate-Early Regulatory Protein, ICP27, Is Responsible for the Aberrant Localization of ICP0 and Mutant Forms of ICP4 in ICP4 Mutant Virus-Infected Cells.

English

Amanote Research

Impaired STING Pathway in Human Osteosarcoma U2OS Cells Contributes to the Growth of ICP0-Null Mutant Herpes Simplex Virus

Journal of Virology - United States

doi 10.1128/jvi.00006-17

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Complementation of a Herpes Simplex Virus ICP0 Null Mutant by Varicella-Zoster Virus ORF61p

Herpes Simplex Virus VP16, but Not ICP0, Is Required to Reduce Histone Occupancy and Enhance Histone Acetylation on Viral Genomes in U2OS Osteosarcoma Cells

An Activity Specified by the Osteosarcoma Line U2OS Can Substitute Functionally for ICP0, a Major Regulatory Protein of Herpes Simplex Virus Type 1.

The Replication Defect of ICP0-Null Mutant Herpes Simplex Virus 1 Can Be Largely Complemented by the Combined Activities of Human Cytomegalovirus Proteins IE1 and Pp71

Ubiquitin-Specific Protease 9X in Host Cells Interacts With Herpes Simplex Virus 1 ICP0

The Null Mutant of the U(L)31 Gene of Herpes Simplex Virus 1: Construction and Phenotype in Infected Cells.

Varicella-Zoster Virus Gene 51 Complements a Herpes Simplex Virus Type 1 UL9 Null Mutant.

Theabrownin Triggers DNA Damage to Suppress Human Osteosarcoma U2OS Cells by Activating P53 Signalling Pathway

Overexpression of the Herpes Simplex Virus Type 1 Immediate-Early Regulatory Protein, ICP27, Is Responsible for the Aberrant Localization of ICP0 and Mutant Forms of ICP4 in ICP4 Mutant Virus-Infected Cells.