(PDF) Chronic Ethanol Feeding Potentiates Fas Jo2-Induced

Chronic Ethanol Feeding Potentiates Fas Jo2-Induced Hepatotoxicity: Role of CYP2E1 and TNF-α and Activation of JNK and P38 MAP Kinase

Free Radical Biology and Medicine - Netherlands

doi 10.1016/j.freeradbiomed.2009.05.021

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September 1, 2009

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Elsevier BV

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Amanote Research

Chronic Ethanol Feeding Potentiates Fas Jo2-Induced Hepatotoxicity: Role of CYP2E1 and TNF-α and Activation of JNK and P38 MAP Kinase

Free Radical Biology and Medicine - Netherlands

doi 10.1016/j.freeradbiomed.2009.05.021

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Genetic Deletion of PKR Abrogates TNF-induced Activation of IκBα Kinase, JNK, Akt and Cell Proliferation but Potentiates P44/P42 MAPK and P38 MAPK Activation

Activation and Signaling of the P38 MAP Kinase Pathway

Role of P38 Mitogen-Activated Protein Kinase Phosphorylation and Fas-Fas Ligand Interaction in Morphine-Induced Macrophage Apoptosis

Mechanism of P38 MAP Kinase Activation in Vivo

SH3BP2 Cherubism Mutation Potentiates TNF-α-Induced Osteoclastogenesis via NFATc1 and TNF-α-Mediated Inflammatory Bone Loss

NF-κB, but Not P38 MAP Kinase, Is Required for TNF-α-induced Expression of Cell Adhesion Molecules in Endothelial Cells

Role of P38 MAP Kinase in LPS-induced Airway Inflammation in the Rat

JNK (C-Jun N-Terminal Kinase) and P38 Activation in Receptor-Mediated and Chemically-Induced Apoptosis of T-Cells: Differential Requirements for Caspase Activation

NDF/heregulin-induced Cell Cycle Changes and Apoptosis in Breast Tumour Cells: Role of PI3 Kinase and P38 MAP Kinase Pathways