Amanote Research
Register
Sign In
Heat-Induced Dimerization of BCL-xL Through A-Helix Swapping
doi 10.1021/bi062080a.s001
Full Text
Open PDF
Abstract
Available in
full text
Date
Unknown
Authors
Unknown
Publisher
American Chemical Society (ACS)
Related search
Modulation of Cell Death by BCL-xL Through Caspase Interaction
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
C-Myc Augments Gamma Irradiation-Induced Apoptosis by Suppressing BCL-Xl
Molecular and Cellular Biology
Cell Biology
Molecular Biology
The BCL-2 Proteins Noxa and BCL-xL Co-Ordinately Regulate Oxidative Stress-Induced Apoptosis
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
BCL-xL in Neuroprotection and Plasticity
Frontiers in Physiology
Physiology
BCL-xL Determines the Metabolic Efficiency of Neurons, Through Interaction With Mitochondrial ATP Synthase
Biophysical Journal
Biophysics
Hill Coefficient Analysis of Transmembrane Helix Dimerization
Journal of Membrane Biology
Cell Biology
Physiology
Biophysics
Structure-Based Discovery of a New Class of BCL-xL Antagonists
Bioorganic Chemistry
Organic Chemistry
Biochemistry
Drug Discovery
Molecular Biology
TNF-α-induced ROS Production Triggering Apoptosis Is Directly Linked to Romo1 and BCL-Xl
Cell Death and Differentiation
Cell Biology
Molecular Biology
HSpin1, a Transmembrane Protein Interacting With BCL-2/BCL-xL, Induces a Caspase-Independent Autophagic Cell Death
Cell Death and Differentiation
Cell Biology
Molecular Biology