BPOZ-2 Gene Delivery Ameliorates Alpha-Synucleinopathy in A53T Transgenic Mouse Model of Parkinson’s Disease

Scientific Reports - United Kingdom
doi 10.1038/srep22067
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

In Vivo Alterations in Calcium Buffering Capacity in Transgenic Mouse Model of Synucleinopathy

Journal of Neuroscience
Neuroscience
2012English

Chemogenetic Modulation of Orexin Neurons Reverses Changes in Anxiety and Locomotor Activity in the A53T Mouse Model of Parkinson’s Disease

Frontiers in Neuroscience
Neuroscience
2019English

Clinical, Neuropathological and Genotypic Variability in SNCA A53T Familial Parkinson’s Disease

Acta Neuropathologica
Forensic MedicineMolecular NeurosciencePathologyNeurologyCellular
2008English

Mitochondrial Division Inhibitor-1 Is Neuroprotective in the A53t-Α-Synuclein Rat Model of Parkinson’s Disease

Scientific Reports
Multidisciplinary
2017English

Corticosterone Dysregulation Exacerbates Disease Progression in the R6/2 Transgenic Mouse Model of Huntington's Disease

Experimental Neurology
Developmental NeuroscienceNeurology
2016English

Neonatal Neurodegeneration in Alzheimer’s Disease Transgenic Mouse Model

Journal of Alzheimer's Disease Reports
2018English

Inactivation of Mouse Alpha-Globin Gene by Homologous Recombination: Mouse Model of Hemoglobin H Disease

Blood
BiochemistryImmunologyCell BiologyHematology
1996English

Knockout-Transgenic Mouse Model of Sickle Cell Disease

Science
MultidisciplinaryPhilosophy of ScienceHistory
1997English

Targeting Isoprenylcysteine Methylation Ameliorates Disease in a Mouse Model of Progeria

Science
MultidisciplinaryPhilosophy of ScienceHistory
2013English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2025 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy