(PDF) NF-κB Hyper-Activation by HTLV-1 Tax Induces Cellular

NF-κB Hyper-Activation by HTLV-1 Tax Induces Cellular Senescence, but Can Be Alleviated by the Viral Anti-Sense Protein HBZ

Retrovirology - United Kingdom

doi 10.1186/1742-4690-8-s1-a200

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January 1, 2011

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Springer Science and Business Media LLC

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NF-κB Hyper-Activation by HTLV-1 Tax Induces Cellular Senescence, but Can Be Alleviated by the Viral Anti-Sense Protein HBZ

Retrovirology - United Kingdom

doi 10.1186/1742-4690-8-s1-a200

Full Text

Abstract

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Date

Authors

Publisher

Related search

Bromodomain and Extraterminal (BET) Protein Inhibition Suppresses Human T Cell Leukemia Virus 1 (HTLV-1) Tax Protein-Mediated Tumorigenesis by Inhibiting Nuclear Factor κB (NF-κB) Signaling

The Tumour Marker Fascin Is Strongly Induced by Tax of HTLV-1 Through NF-κB Signals in T Lymphocytes

HTLV-1 Tax-Activated NF-κB Is Involved in Both Cell Growth and Death

Exosomes Derived From HTLV-1 Infected Cells Contain the Viral Protein Tax

HTLV-1 HBZ Protein Deregulates Interactions Between Cellular Factors and the KIX Domain of P300/CBP

Stability of the HTLV-1 Antisense-Derived Protein, HBZ, Is Regulated by the E3 Ubiquitin-Protein Ligase, UBR5

P53 Induces NF-κB Activation by an IκB Kinase-Independent Mechanism Involving Phosphorylation of P65 by Ribosomal S6 Kinase 1

HTLV-1 Tax-Induced Rapid Senescence Is Driven by the Transcriptional Activity of NF- B and Depends on Chronically Activated IKK and P65/RelA

Honokiol Stimulates Osteoblastogenesis by Suppressing NF-κB Activation