High Expression of SLC26A6 in the Kidney May Contribute to Renal Calcification via an SLC26A6-dependent Mechanism

PeerJ - United States
doi 10.7717/peerj.5192
Full Text
Abstract

Available in full text

Date
Authors
Publisher

PeerJ

Related search

N -Glycosylation Critically Regulates Function of Oxalate Transporter SLC26A6

American Journal of Physiology - Cell Physiology
PhysiologyCell Biology
2016English

SLC26A6 and NaDC-1 Transporters Interact to Regulate Oxalate and Citrate Homeostasis

Journal of the American Society of Nephrology : JASN
MedicineNephrology
2013English

High-Dose Estradiol-Replacement Therapy Enhances the Renal Vascular Response to Angiotensin II via an AT2-Receptor Dependent Mechanism

Advances in Pharmacological Sciences
ToxicologyMolecular MedicinePharmaceuticsPharmacology
2015English

Involvement of Cl−/Hco3− Exchanger SLC26A3 and SLC26A6 in Preimplantation Embryo Cleavage

Scientific Reports
Multidisciplinary
2016English

Isoforms of SLC26A6 Mediate Anion Transport and Have Functional PDZ Interaction Domains

American Journal of Physiology - Cell Physiology
PhysiologyCell Biology
2003English

Novel Frataxin Isoforms May Contribute to the Pathological Mechanism of Friedreich Ataxia

PLoS ONE
Multidisciplinary
2012English

Biophysical Stimulation Induces Demyelination via an Integrin-Dependent Mechanism

Annals of Neurology
Neurology
2012English

Estrogen Inhibits Vascular Calcification via Vascular RANKL System: Common Mechanism of Osteoporosis and Vascular Calcification

Circulation Research
Cardiovascular MedicinePhysiologyCardiology
2010English

To Save or Not to Save the Kidney: Relieving Unilateral Obstruction May Significantly Improve an Initially Low Split Renal Creatinine Clearance

Journal of Urological Surgery
2016English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy