Nitric Oxide Signaling Is Recruited as a Compensatory Mechanism for Sustaining Synaptic Plasticity in Alzheimer's Disease Mice

Journal of Neuroscience - United States
doi 10.1523/jneurosci.4002-14.2015
Full Text
Abstract

Available in full text

Categories
Neuroscience
Date
Authors
Publisher

Society for Neuroscience

Related search

Early Presynaptic and Postsynaptic Calcium Signaling Abnormalities Mask Underlying Synaptic Depression in Presymptomatic Alzheimer's Disease Mice

Journal of Neuroscience
Neuroscience
2012English

Erratum To: Nitric Oxide Signaling in Health and Disease

2017English

Synaptic Plasticity in Alzheimer’s Disease

Revista Neurociências
2001English

Endothelial Nitric Oxide Signaling Regulates Notch1 in Aortic Valve Disease

Journal of Molecular and Cellular Cardiology
Cardiovascular MedicineMolecular BiologyCardiology
2013English

Nitric Oxide Signaling in Plants

Frontiers in Plant Science
Plant Science
2014English

Synaptic Compensatory Mechanism and Its Impairment in Autoimmune Myasthenic Diseases

Journal of Immunological Sciences
2019English

Local Signaling With Molecular Diffusion as a Decoder of Ca2+ Signals in Synaptic Plasticity

Molecular Systems Biology
Information SystemsGeneticsMolecular BiologyBiochemistryApplied MathematicsComputational TheoryMathematicsMicrobiology ImmunologyBiological SciencesMedicineAgricultural
2005English

Glucagon-Like Peptide-1 Cleavage Product GLP-1(9-36) Amide Rescues Synaptic Plasticity and Memory Deficits in Alzheimer's Disease Model Mice

Journal of Neuroscience
Neuroscience
2012English

Macrophage Nitric Oxide Synthase Associates With Cortical Actin but Is Not Recruited to Phagosomes

Infection and Immunity
ParasitologyInfectious DiseasesImmunologyMicrobiology
2001English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy