TAK1 Restricts Spontaneous NLRP3 Activation and Cell Death to Control Myeloid Proliferation

Journal of Experimental Medicine - United States
doi 10.1084/jem.20171922
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Rockefeller University Press

Related search

TAK1 Regulates Caspase 8 Activation and Necroptotic Signaling via Multiple Cell Death Checkpoints

Cell Death and Disease
Molecular NeuroscienceImmunologyCell BiologyCancer ResearchCellularMedicine
2016English

TAK1 Activation of Alpha-Tat1 and Microtubule Hyperacetylation Control AKT Signaling and Cell Growth

Nature Communications
AstronomyGeneticsMolecular BiologyBiochemistryChemistryPhysics
2018English

Programmed Cell Death Ligand 1 (PD-L1) Signaling Regulates Macrophage Proliferation and Activation

Cancer immunology research
Cancer ResearchImmunology
2018English

Retinal Pigment Epithelium Cell Death Is Associated With NLRP3 Inflammasome Activation by All-Trans Retinal

Investigative Ophthalmology and Visual Science
Molecular NeuroscienceOphthalmologySensory SystemsCellular
2019English

Deletion of TAK1 in the Myeloid Lineage Results in the Spontaneous Development of Myelomonocytic Leukemia in Mice

PLoS ONE
Multidisciplinary
2012English

Linking Cancer-Induced Nlrp3 Inflammasome Activation to Efficient NK Cell-Mediated Immunosurveillance

OncoImmunology
OncologyAllergyImmunology
2016English

Mebendazole Stimulates CD14+ Myeloid Cells to Enhance T-Cell Activation and Tumour Cell Killing

Oncotarget
Oncology
2018English

NLRP12 Collaborates With NLRP3 and NLRC4 to Promote Pyroptosis Inducing Ganglion Cell Death of Acute Glaucoma

Molecular Neurodegeneration
Molecular NeuroscienceNeurologyMolecular BiologyCellular
2020English

OR11-004 - IL-1, IL-18 and Cell Death in NLRP3 Driven Disease

Pediatric Rheumatology
ImmunologyPediatricsRheumatologyAllergyPerinatologyChild Health
2013English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy