(PDF) C-Myc Sensitizes Cells to Tumor Necrosis

C-Myc Sensitizes Cells to Tumor Necrosis Factor-Mediated Apoptosis by Inhibiting Nuclear Factor κB Transactivation

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m203213200

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July 30, 2002

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American Society for Biochemistry & Molecular Biology (ASBMB)

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Amanote Research

C-Myc Sensitizes Cells to Tumor Necrosis Factor-Mediated Apoptosis by Inhibiting Nuclear Factor κB Transactivation

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m203213200

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Tumor Necrosis Factor-Α Induces Transcriptional Activation of Nuclear Factor-κB in Insulin-Producing Β-Cells

SIMPL Is a Tumor Necrosis Factor-Specific Regulator of Nuclear Factor-κB Activity

Zoledronate Sensitizes Endothelial Cells to Tumor Necrosis Factor-Induced Programmed Cell Death

Expression of Nuclear Factor-ΚB, Tumor Necrosis Factor Receptor Type 1, and C-Myc in Human Astrocytomas

The Tumor Suppressor CYLD Interacts With TRIP and Regulates Negatively Nuclear Factor κB Activation by Tumor Necrosis Factor

Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand–Mediated Proliferation of Tumor Cells With Receptor-Proximal Apoptosis Defects

Gliotoxin Potentiates Osteoblast Differentiation by Inhibiting Nuclear Factor-κB Signaling

LIGHT, a Member of the Tumor Necrosis Factor Ligand Superfamily, Prevents Tumor Necrosis Factor-Α-Mediated Human Primary Hepatocyte Apoptosis, but Not Fas-Mediated Apoptosis

Amanote Research

C-Myc Sensitizes Cells to Tumor Necrosis Factor-Mediated Apoptosis by Inhibiting Nuclear Factor κB Transactivation

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m203213200

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Tumor Necrosis Factor-Α Induces Transcriptional Activation of Nuclear Factor-κB in Insulin-Producing Β-Cells

SIMPL Is a Tumor Necrosis Factor-Specific Regulator of Nuclear Factor-κB Activity

Zoledronate Sensitizes Endothelial Cells to Tumor Necrosis Factor-Induced Programmed Cell Death

Expression of Nuclear Factor-&Kappa;B, Tumor Necrosis Factor Receptor Type 1, and C-Myc in Human Astrocytomas

The Tumor Suppressor CYLD Interacts With TRIP and Regulates Negatively Nuclear Factor κB Activation by Tumor Necrosis Factor

Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand–Mediated Proliferation of Tumor Cells With Receptor-Proximal Apoptosis Defects

Gliotoxin Potentiates Osteoblast Differentiation by Inhibiting Nuclear Factor-κB Signaling

LIGHT, a Member of the Tumor Necrosis Factor Ligand Superfamily, Prevents Tumor Necrosis Factor-Α-Mediated Human Primary Hepatocyte Apoptosis, but Not Fas-Mediated Apoptosis

Expression of Nuclear Factor-ΚB, Tumor Necrosis Factor Receptor Type 1, and C-Myc in Human Astrocytomas