(PDF) Identification of a U5-Specific Sequence Required for

Identification of a U5-Specific Sequence Required for Efficient Polyadenylation Within the Human Immunodeficiency Virus Long Terminal Repeat.

Journal of Virology - United States

doi 10.1128/jvi.63.1.421-424.1989

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Abstract

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Date

January 1, 1989

Authors

S Böhnlein

J Hauber

B R Cullen

Publisher

American Society for Microbiology

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Amanote Research

Identification of a U5-Specific Sequence Required for Efficient Polyadenylation Within the Human Immunodeficiency Virus Long Terminal Repeat.

Journal of Virology - United States

doi 10.1128/jvi.63.1.421-424.1989

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Identification of Amino Acids in HIV-1 and Avian Sarcoma Virus Integrase Subsites Required for Specific Recognition of the Long Terminal Repeat Ends

Nucleotide Sequence Analysis of the Long Terminal Repeat of Human T-Cell Leukemia Virus Type II.

Murine Leukemia Virus-Based Tat-Inducible Long Terminal Repeat Replacement Vectors: A New System for Anti-Human Immunodeficiency Virus Gene Therapy.

Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

Nucleotide Substitutions Within U5 Are Critical for Efficient Reverse Transcription of Human Immunodeficiency Virus Type 1 With a Primer Binding Site Complementary to tRNA(His).

The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1

Sequence of the Long Terminal Repeat and Adjacent Segments of the Endogenous Avian Virus Rous-Associated Virus 0

Human Immunodeficiency Virus Type 1 Long Terminal Repeat Variants From 42 Patients Representing All Stages of Infection Display a Wide Range of Sequence Polymorphism and Transcription Activity.

Stimulation of Tat-Associated Kinase-Independent Transcriptional Elongation From the Human Immunodeficiency Virus Type-1 Long Terminal Repeat by a Cellular Enhancer