Β-Arrestin–biased Signaling Mediates Memory Reconsolidation

Proceedings of the National Academy of Sciences of the United States of America - United States
doi 10.1073/pnas.1421758112
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Proceedings of the National Academy of Sciences

Related search

Molecular Mechanisms of Fentanyl Mediated Β-Arrestin Biased Signaling

2019English

Β-Arrestin–biased Signaling Through the Β2-Adrenergic Receptor Promotes Cardiomyocyte Contraction

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2016English

Memory Reconsolidation Mediates the Updating of Hippocampal Memory Content

Frontiers in Behavioral Neuroscience
Cognitive NeurosciencePhysiological PsychologyBehavioral NeuroscienceNeuropsychology
2010English

On the Involvement of BDNF Signaling in Memory Reconsolidation

Frontiers in Cellular Neuroscience
Molecular NeuroscienceCellular
2019English

Β-Arrestin Mediates the Frank–Starling Mechanism of Cardiac Contractility

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2016English

Characterization of Methadone as a Β-Arrestin-Biased Μ-Opioid Receptor Agonist

Molecular Pain
Molecular MedicineAnesthesiologyPain MedicineCellularMolecular Neuroscience
2016English

Beta-Arrestin Biased Signaling at a Class a GPCR: Modeling the ORG27569 Induced CB1/Beta-Arrestin 1 Complex

Biophysical Journal
Biophysics
2015English

Β-Arrestin-Biased Agonists of the GLP-1 Receptor From Β-Amino Acid Residue Incorporation Into GLP-1 Analogues

Journal of the American Chemical Society
BiochemistryColloidCatalysisChemistrySurface Chemistry
2016English

Acute Exercise on Memory Reconsolidation

Journal of Clinical Medicine
2019English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy