The Chimeric Genes AML1/MDS1 and AML1/EAP Inhibit AML1B Activation at the CSF1R Promoter, but Only AML1/MDS1 Has Tumor-Promoter Properties.

Proceedings of the National Academy of Sciences of the United States of America- United States

doi 10.1073/pnas.93.3.1044

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February 6, 1996

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Proceedings of the National Academy of Sciences

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The Chimeric Genes AML1/MDS1 and AML1/EAP Inhibit AML1B Activation at the CSF1R Promoter, but Only AML1/MDS1 Has Tumor-Promoter Properties.

Proceedings of the National Academy of Sciences of the United States of America- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Targeted Degradation of the AML1/MDS1/EVI1 Oncoprotein by Arsenic Trioxide

AML1 (CBFα2) Cooperates With B Cell-Specific Activating Protein (BSAP/PAX5) in Activation of the B Cell-specificBLKGene Promoter

Three Distinct Domains in TEL-AML1 Are Required for Transcriptional Repression of the IL-3 Promoter

AML1 (Acute Myeloid Leukemia 1)

CCAAT Enhancer-Binding Protein (C/Ebp) and AML1 (CBF Alpha2) Synergistically Activate the Macrophage Colony-Stimulating Factor Receptor Promoter.

Acute Myeloid Leukemia With T(3;21)(q26.2;q22) Developing Following Low‑dose Methotrexate Therapy for Rheumatoid Arthritis and Expressing Two AML1/MDS1/EVI1 Fusion Proteins: A Case Report

Epigenetic Consequences of AML1-ETO Action at the Human C-FMS Locus

Significance of the TEL-AML1 Fusion Gene in Childhood AML

Detection of DNA Rearrangements in the AML1 and ETO Loci and of an AML1/ETO Fusion mRNA in Patients With T(8;21) Acute Myeloid Leukemia