Suppression of Senescence in Normal Human Fibroblasts by Introduction of Dominant-Negative P53 Mutants or Human Papilloma Virus Type 16 E6 Protein

Bioscience, Biotechnology and Biochemistry - United Kingdom
doi 10.1271/bbb.62.1458
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Informa UK Limited

Related search

Human Papilloma Virus 16 E6 Oncoprotein Inhibits Retinoic X Receptor-Mediated Transactivation by Targeting Human ADA3 Coactivator

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2002English

Role of IFI 16 in Cellular Senescence of Human Fibroblasts

Oncogene
Cancer ResearchGeneticsMolecular Biology
2004English

Oligonucleotide Array Analysis of Telomerase Activation by the Human Papillomavirus Type 16 E6 Protein

Nature Genetics
Genetics
2001English

Degradation of P53, Not Telomerase Activation, by E6 Is Required for Bypass of Crisis and Immortalization by Human Papillomavirus Type 16 E6/E7

Journal of Virology
Insect ScienceImmunologyMicrobiologyVirology
2004English

Knockdown of CDK2AP1 in Primary Human Fibroblasts Induces P53 Dependent Senescence

PLoS ONE
Multidisciplinary
2015English

The ING4 Binding With P53 and Induced P53 Acetylation Were Attenuated by Human Papillomavirus 16 E6

PLoS ONE
Multidisciplinary
2013English

Knockdown of MBP-1 in Human Foreskin Fibroblasts Induces P53-P21 Dependent Senescence

PLoS ONE
Multidisciplinary
2008English

Reversible Cellular Senescence: Implications for Immortalization of Normal Human Diploid Fibroblasts.

Molecular and Cellular Biology
Cell BiologyMolecular Biology
1989English

Chromosomal Instability in Human Mesenchymal Stem Cells Immortalized With Human Papilloma Virus E6, E7, and hTERT Genes

In Vitro Cellular and Developmental Biology - Animal
MedicineDevelopmental BiologyCell Biology
2007English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy