(PDF) AKIP1 Enhances NF-κB-dependent Gene Expression by

AKIP1 Enhances NF-κB-dependent Gene Expression by Promoting the Nuclear Retention and Phosphorylation of P65

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m710285200

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January 4, 2008

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American Society for Biochemistry & Molecular Biology (ASBMB)

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AKIP1 Enhances NF-κB-dependent Gene Expression by Promoting the Nuclear Retention and Phosphorylation of P65

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m710285200

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Casein Kinase II-mediated Phosphorylation of NF-κB P65 Subunit Enhances Inducible Nitric-Oxide Synthase Gene Transcriptionin Vivo

Site-Specific Phosphorylation of the P65 Protein Subunit Mediates Selective Gene Expression by Differential NF-κB and RNA Polymerase II Promoter Recruitment

Retraction: Modification of Cysteine Residue in P65 Subunit of Nuclear Factor-κB (NF-κB) by Picroliv Suppresses NF-κB–Regulated Gene Products and Potentiates Apoptosis

TDP-43 Inhibits NF-κB Activity by Blocking P65 Nuclear Translocation

Fas-Associated Factor-1 Inhibits Nuclear Factor-κB (NF-κB) Activity by Interfering With Nuclear Translocation of the RelA (P65) Subunit of NF-κB

SIRT2 Regulates NF- B-Dependent Gene Expression Through Deacetylation of P65 Lys310

Transactivation of Gene Expression by NF-κB Is Dependent on Thioredoxin Reductase Activity

Inducible Phosphorylation of NF-κB P65 at Serine 468 by T Cell Costimulation Is Mediated by IKKϵ

Chemotherapeutic Drugs Inhibiting Topoisomerase 1 Activity Impede Cytokine-Induced and NF-κB P65-Regulated Gene Expression