(PDF) Expression of TLR4-MyD88 and NF-κB in the Iris During

Expression of TLR4-MyD88 and NF-κB in the Iris During Endotoxin-Induced Uveitis

Mediators of Inflammation - Egypt

doi 10.1155/2010/748218

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January 1, 2010

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Hindawi Limited

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Expression of TLR4-MyD88 and NF-κB in the Iris During Endotoxin-Induced Uveitis

Mediators of Inflammation - Egypt

doi 10.1155/2010/748218

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

NF-κB Inhibition Attenuates LPS-induced TLR4 Activation in Monocyte Cells

Aloin Preconditioning Attenuates Hepatic Ischemia/Reperfusion Injury via Inhibiting TLR4/MyD88/NF-κB Signal Pathway in Vivo and in Vitro

Endotoxin Stress-Response in Cardiomyocytes: NF-κB Activation and Tumor Necrosis Factor-Α Expression

Activation of Vascular Endothelial Growth Factor Receptor-3 in Macrophages Restrains TLR4-NF-κB Signaling and Protects Against Endotoxin Shock

Baicalein Inhibits TNF-α-induced NF-κB Activation and Expression of NF-κB-regulated Target Gene Products

Endotoxin-Induced Maturation of MyD88-Deficient Dendritic Cells

NF-κB Mediates Tumor Necrosis Factor Α-Induced Expression of Optineurin, a Negative Regulator of NF-κB

LPS Enhances TLR4 Expression and IFN-γ Production via the TLR4/IRAK/NF-κB Signaling Pathway in Rat Pulmonary Arterial Smooth Muscle Cells

Boosting mTOR-dependent Autophagy via Upstream TLR4-MyD88-MAPK Signalling and Downstream NF-κB Pathway Quenches Intestinal Inflammation and Oxidative Stress Injury