(PDF) Human Memory FOXP3+ Tregs Secrete IL-17 Ex Vivo and

Human Memory FOXP3+ Tregs Secrete IL-17 Ex Vivo and Constitutively Express the TH17 Lineage-Specific Transcription Factor ROR T

Proceedings of the National Academy of Sciences of the United States of America - United States

doi 10.1073/pnas.0900621106

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May 13, 2009

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Proceedings of the National Academy of Sciences

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Human Memory FOXP3+ Tregs Secrete IL-17 Ex Vivo and Constitutively Express the TH17 Lineage-Specific Transcription Factor ROR T

Proceedings of the National Academy of Sciences of the United States of America - United States

doi 10.1073/pnas.0900621106

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Th17 Differentiation in Hyper IgE Syndrome; IL-17 Secretion and RORγt Expression

Cutting Edge: NKT Cells Constitutively Express IL-23 Receptor and ROR T and Rapidly Produce IL-17 Upon Receptor Ligation in an IL-6-Independent Fashion

Transcription Factor KLF10 Constrains IL-17-Committed Vγ4+ Γδ T Cells

Exogenous IFN-γ Ex Vivo Shapes the Alloreactive T-Cell Repertoire by Inhibition of Th17 Responses and Generation of Functional Foxp3+ Regulatory T Cells

The IL23R a/Gln381 Allele Promotes IL-23 Unresponsiveness in Human Memory T-Helper 17 Cells and Impairs Th17 Responses in Psoriasis Patients

Ex Vivo Expansion of CD4+CD25+FoxP3+ T Regulatory Cells Based on Synergy Between IL-2 and 4-1bb Signaling

Th17 Cells Express Interleukin-10 Receptor and Are Controlled by Foxp3− and Foxp3+ Regulatory CD4+ T Cells in an Interleukin-10-Dependent Manner

Leprosy Reactions Show Increased Th17 Cell Activity and Reduced FOXP3+ Tregs With Concomitant Decrease in TGF-β and Increase in IL-6

Lineage-Specific and Ubiquitous Biological Roles of the Mammalian Transcription Factor LSF

Amanote Research

Human Memory FOXP3+ Tregs Secrete IL-17 Ex Vivo and Constitutively Express the TH17 Lineage-Specific Transcription Factor ROR T

Proceedings of the National Academy of Sciences of the United States of America - United States

doi 10.1073/pnas.0900621106

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Th17 Differentiation in Hyper IgE Syndrome; IL-17 Secretion and ROR&#947;t Expression

Cutting Edge: NKT Cells Constitutively Express IL-23 Receptor and ROR T and Rapidly Produce IL-17 Upon Receptor Ligation in an IL-6-Independent Fashion

Transcription Factor KLF10 Constrains IL-17-Committed Vγ4+ Γδ T Cells

Exogenous IFN-γ Ex Vivo Shapes the Alloreactive T-Cell Repertoire by Inhibition of Th17 Responses and Generation of Functional Foxp3+ Regulatory T Cells

The IL23R a/Gln381 Allele Promotes IL-23 Unresponsiveness in Human Memory T-Helper 17 Cells and Impairs Th17 Responses in Psoriasis Patients

Ex Vivo Expansion of CD4+CD25+FoxP3+ T Regulatory Cells Based on Synergy Between IL-2 and 4-1bb Signaling

Th17 Cells Express Interleukin-10 Receptor and Are Controlled by Foxp3− and Foxp3+ Regulatory CD4+ T Cells in an Interleukin-10-Dependent Manner

Leprosy Reactions Show Increased Th17 Cell Activity and Reduced FOXP3+ Tregs With Concomitant Decrease in TGF-β and Increase in IL-6

Lineage-Specific and Ubiquitous Biological Roles of the Mammalian Transcription Factor LSF

Th17 Differentiation in Hyper IgE Syndrome; IL-17 Secretion and RORγt Expression