(PDF) Erratum to “Palmitic Acid Induces Production of

Erratum to “Palmitic Acid Induces Production of Proinflammatory Cytokines Interleukin-6, Interleukin-1β, and Tumor Necrosis Factor-Αvia a NF-κB-Dependent Mechanism in HaCaT Keratinocytes”

Mediators of Inflammation - Egypt

doi 10.1155/2014/513027

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January 1, 2014

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Hindawi Limited

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Erratum to “Palmitic Acid Induces Production of Proinflammatory Cytokines Interleukin-6, Interleukin-1β, and Tumor Necrosis Factor-Αvia a NF-κB-Dependent Mechanism in HaCaT Keratinocytes”

Mediators of Inflammation - Egypt

doi 10.1155/2014/513027

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Interleukin-17 Induces Hyperresponsive Interleukin-8 and Interleukin-6 Production to Tumor Necrosis Factor-Α in Structural Lung Cells

Production of Tumor Necrosis Factor Alpha, Interleukin-1 Alpha, and Interleukin-6 During Murine Coccidioidomycosis.

NF-κB; IL-1β; Tumor Necrosis Factor-Α (TNF-α)

Suppression of Proinflammatory Cytokines Interleukin-1 and Tumor Necrosis Factor- In Astrocytes by a V1 Vasopressin Receptor Agonist: A cAMP Response Element-Binding Protein-Dependent Mechanism

Polyamine Depletion Inhibits NF-κB Binding to DNA and Interleukin-8 Production in Human Chondrocytes Stimulated by Tumor Necrosis Factor-Α

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Tumor Necrosis Factor-Α and Interleukin-1β Regulate Interleukin-8 Expression in Third Trimester Decidual Cells

Racial Differences in Tumor Necrosis Factor-Α-Induced Endothelial Microparticles and Interleukin-6 Production

Curcumin Derivative, 2,6-Bis(2-Fluorobenzylidene)cyclohexanone (MS65) Inhibits Interleukin-6 Production Through Suppression of NF-κB and MAPK Pathways in Histamine-Induced Human Keratinocytes Cell (HaCaT)

Amanote Research

Erratum to “Palmitic Acid Induces Production of Proinflammatory Cytokines Interleukin-6, Interleukin-1β, and Tumor Necrosis Factor-Αvia a NF-κB-Dependent Mechanism in HaCaT Keratinocytes”

Mediators of Inflammation - Egypt

doi 10.1155/2014/513027

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Interleukin-17 Induces Hyperresponsive Interleukin-8 and Interleukin-6 Production to Tumor Necrosis Factor-Α in Structural Lung Cells

Production of Tumor Necrosis Factor Alpha, Interleukin-1 Alpha, and Interleukin-6 During Murine Coccidioidomycosis.

NF-κB; IL-1β; Tumor Necrosis Factor-Α (TNF-α)

Suppression of Proinflammatory Cytokines Interleukin-1 and Tumor Necrosis Factor- In Astrocytes by a V1 Vasopressin Receptor Agonist: A cAMP Response Element-Binding Protein-Dependent Mechanism

Polyamine Depletion Inhibits NF-κB Binding to DNA and Interleukin-8 Production in Human Chondrocytes Stimulated by Tumor Necrosis Factor-Α

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Tumor Necrosis Factor-Α and Interleukin-1β Regulate Interleukin-8 Expression in Third Trimester Decidual Cells

Racial Differences in Tumor Necrosis Factor-&Alpha;-Induced Endothelial Microparticles and Interleukin-6 Production

Curcumin Derivative, 2,6-Bis(2-Fluorobenzylidene)cyclohexanone (MS65) Inhibits Interleukin-6 Production Through Suppression of NF-κB and MAPK Pathways in Histamine-Induced Human Keratinocytes Cell (HaCaT)

Racial Differences in Tumor Necrosis Factor-Α-Induced Endothelial Microparticles and Interleukin-6 Production