(PDF) Protein Tyrosine Kinase P56lckIs Required for

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Journal of Biological Chemistry - United States

doi 10.1074/jbc.275.18.13297

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Date

April 28, 2000

Authors

Sunil K. Manna

Nand K. Sah

Bharat B. Aggarwal

Publisher

American Society for Biochemistry & Molecular Biology (ASBMB)

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Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Journal of Biological Chemistry - United States

doi 10.1074/jbc.275.18.13297

Full Text

Abstract

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Date

Authors

Publisher

Related search

Tumor Necrosis Factor-Regulated Biphasic Activation of NF-κB Is Required for Cytokine-Induced Loss of Skeletal Muscle Gene Products

Functional P53 Is Required for Triptolide-Induced Apoptosis and AP-1 and Nuclear Factor-κB Activation in Gastric Cancer Cells

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

NF-κB Mediates Tumor Necrosis Factor Α-Induced Expression of Optineurin, a Negative Regulator of NF-κB

Cross-Talk Between Receptor Tyrosine Kinase and Tumor Necrosis Factor-Α Signaling Networks Regulates Apoptosis but Not Proliferation

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells