Modulation of 3-Methylcholanthrene Toxicity in Cultured Neoplastic Keratinocytes by Glucocorticoids and Retinoids Is Not Accounted for by Macromolecular Adduct Formation.

Proceedings of the National Academy of Sciences of the United States of America- United States

doi 10.1073/pnas.86.8.2961

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April 1, 1989

Authors

A. L. Rubin

R. H. Rice

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Proceedings of the National Academy of Sciences

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Modulation of 3-Methylcholanthrene Toxicity in Cultured Neoplastic Keratinocytes by Glucocorticoids and Retinoids Is Not Accounted for by Macromolecular Adduct Formation.

Proceedings of the National Academy of Sciences of the United States of America- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Differential Toxicity Response of Normal and Neoplastic Cells in Vitro to 3,4-Benzopyrene and 3-Methylcholanthrene

Impact of Genetic Modulation of SULT1A Enzymes on DNA Adduct Formation by Aristolochic Acids and 3-Nitrobenzanthrone

Cornifin, a Cross-Linked Envelope Precursor in Keratinocytes That Is Down-Regulated by Retinoids.

Omega-3 Polyunsaturated Fatty Acids Selectively Inhibit Growth in Neoplastic Oral Keratinocytes by Differentially Activating ERK1/2

Protein Targets of Acrylamide Adduct Formation in Cultured Rat Dopaminergic Cells

Modulation of Collagen Synthesis and Degradation by Retinoids and Cytokines in 3T3 L1 Preadipocytes.

Cell Formation by Myxozoan Species Is Not Explained by Dogma

Arsine Toxicity Is Induced by Inhalation but Not by Percutaneous Exposure in Hairless Mice

Intestinal Trefoil Factor Gene Expression in Intestinal Epithelial Cells Is Not Regulated by Glucocorticoids