Plasmin- And Thrombin-Accelerated Shedding of Syndecan-4 Ectodomain Generates Cleavage Sites at Lys114–Arg115and Lys129–Val130Bonds

Journal of Biological Chemistry - United States
doi 10.1074/jbc.m501903200
Full Text
Abstract

Available in full text

Date
Authors
Publisher

American Society for Biochemistry & Molecular Biology (ASBMB)

Related search

Activation of Syndecan-1 Ectodomain Shedding byStaphylococcus Aureusα-Toxin and Β-Toxin

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2003English

Regulated Shedding of Syndecan-1 and -4 Ectodomains by Thrombin and Growth Factor Receptor Activation

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1997English

Ectodomain Shedding and Intramembrane Cleavage of Mammalian Notch Proteins Are Not Regulated Through Oligomerization

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2004English

Matrix Metalloproteinase Mediated Shedding of Syndecan-4 Under Osteoarthritis Conditions

Osteoarthritis and Cartilage
Sports MedicineRheumatologyOrthopedicsBiomedical Engineering
2019English

Protease-Mediated Ectodomain Shedding

Thorax
PulmonaryRespiratory Medicine
2013English

Interspecies Comparison of Simultaneous Thrombin and Plasmin Generation

Scientific Reports
Multidisciplinary
2020English

Regulated Shedding of Syndecan Ectodomains by Chemokines

The Scientific World Journal
BiochemistryMedicineGeneticsMolecular BiologyEnvironmental Science
2006English

Syndecan-4 Is Required for Thrombin-Induced Migration and Proliferation in Human Vascular Smooth Muscle Cells

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2005English

Syndecan 1 Shedding Contributes to Pseudomonas Aeruginosa Sepsis

Infection and Immunity
ParasitologyInfectious DiseasesImmunologyMicrobiology
2005English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy