P2-Purinoceptor-Mediated Formation of Inositol Phosphates and Intracellular Ca2+ Transients in Human Coronary Artery Smooth Muscle Cells

British Journal of Pharmacology- United States

doi 10.1111/j.1476-5381.1996.tb15587.x

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August 1, 1996

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Wiley

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P2-Purinoceptor-Mediated Formation of Inositol Phosphates and Intracellular Ca2+ Transients in Human Coronary Artery Smooth Muscle Cells

British Journal of Pharmacology- United States

Full Text

Abstract

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Date

Authors

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Related search

Multiple Effects of Ryanodine on Intracellular Free Ca2+ in Smooth Muscle Cells From Bovine and Porcine Coronary Artery: Modulation of Sarcoplasmic Reticulum Function

Regulation of Ca2+ Influx in Myeloid Cells. Role of Plasma Membrane Potential, Inositol Phosphates, Cytosolic Free [Ca2+], and Filling State of Intracellular Ca2+ Stores.

Intracellular Ca2+ Handling in Vascular Smooth Muscle Cells Is Affected by Proliferation

NO Hyperpolarizes Pulmonary Artery Smooth Muscle Cells and Decreases the Intracellular Ca2+ Concentration by Activating Voltage-Gated K+ Channels.

Increases in Intracellular Calcium via Activation of an Endogenous P2-Purinoceptor in Cultured CHO-K1 Cells

Stimulatory Interactions Between Human Coronary Smooth Muscle Cells and Dendritic Cells

Pharmacology of [3h]-Pyrilamine Binding and of the Histamine-Induced Inositol Phosphates Generation, Intracellular Ca2+-Mobilization and Cytokine Release From Human Corneal Epithelial Cells

Increased Endothelin-1 Responsiveness in Human Coronary Artery Smooth Muscle Cells Exposed to 1,25-Dihydroxyvitamin D3

Regulation of Ryanodine Receptor–Mediated Ca2+ Release in Vas Deferens Smooth Muscle Cells