(PDF) Involvement of Long Terminal Repeat U3 Sequences

Involvement of Long Terminal Repeat U3 Sequences Overlapping the Transcription Control Region in Human Immunodeficiency Virus Type 1 mRNA 3' End Formation.

Molecular and Cellular Biology - United States

doi 10.1128/mcb.11.3.1624

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Abstract

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Date

March 1, 1991

Authors

J D DeZazzo

J E Kilpatrick

M J Imperiale

Publisher

American Society for Microbiology

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Sequences in the Human Immunodeficiency Virus Type 1 U3 Region Required for in Vivo and in Vitro Integration.

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Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

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Repression in Vitro, by Human Adenovirus E1A Protein Domains, of Basal or Tat-Activated Transcription of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat.

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The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1

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Sequences in the 5' and 3' R Elements of Human Immunodeficiency Virus Type 1 Critical for Efficient Reverse Transcription

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Interactions of Thyroid Hormone Receptor With the Human Immunodeficiency Virus Type 1 (HIV-1) Long Terminal Repeat and the HIV-1 Tat Transactivator.

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Blockade of Human Immunodeficiency Virus Type 1 Production in CD4+ T Cells by an Intracellular CD4 Expressed Under Control of the Viral Long Terminal Repeat.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1993

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Cooperation Between Herpes Simplex Virus Type 1-Encoded ICP0 and Tat to Support Transcription of Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Vivo Can Occur in the Absence of the TAR Binding Site.

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Epstein-Barr Virus Latent Membrane Protein Transactivates the Human Immunodeficiency Virus Type 1 Long Terminal Repeat Through Induction of NF-kappa B Activity.

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Amanote Research

Involvement of Long Terminal Repeat U3 Sequences Overlapping the Transcription Control Region in Human Immunodeficiency Virus Type 1 mRNA 3' End Formation.

Molecular and Cellular Biology - United States

doi 10.1128/mcb.11.3.1624

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Sequences in the Human Immunodeficiency Virus Type 1 U3 Region Required for in Vivo and in Vitro Integration.

Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

Repression in Vitro, by Human Adenovirus E1A Protein Domains, of Basal or Tat-Activated Transcription of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat.

The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1

Sequences in the 5' and 3' R Elements of Human Immunodeficiency Virus Type 1 Critical for Efficient Reverse Transcription

Interactions of Thyroid Hormone Receptor With the Human Immunodeficiency Virus Type 1 (HIV-1) Long Terminal Repeat and the HIV-1 Tat Transactivator.

Blockade of Human Immunodeficiency Virus Type 1 Production in CD4+ T Cells by an Intracellular CD4 Expressed Under Control of the Viral Long Terminal Repeat.

Cooperation Between Herpes Simplex Virus Type 1-Encoded ICP0 and Tat to Support Transcription of Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Vivo Can Occur in the Absence of the TAR Binding Site.

Epstein-Barr Virus Latent Membrane Protein Transactivates the Human Immunodeficiency Virus Type 1 Long Terminal Repeat Through Induction of NF-kappa B Activity.