A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Biophysical Journal - United States
doi 10.1016/j.bpj.2018.11.629
Full Text
Abstract

Available in full text

Categories
Biophysics
Date
Authors
Publisher

Elsevier BV

Related search

Fenestrations Control the Resting State Block of a Voltage Gated Sodium Channel

Biophysical Journal
Biophysics
2019English

KCNE3 Stabilizes the Voltage Sensor S4 of KCNQ1 Channel, KCNE1 Uncouples S4 and the Gate

Biophysical Journal
Biophysics
2015English

The Tarantula Toxin Guangxitoxin-1e Traps K + Channel Voltage Sensor in a Fully Resting Conformation

Biophysical Journal
Biophysics
2017English

Voltage-Sensing Residues in the Voltage Sensor of the BK Channel

Biophysical Journal
Biophysics
2019English

Myokymia and Neonatal Epilepsy Caused by a Mutation in the Voltage Sensor of the KCNQ2 K+ Channel

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2001English

Structure of the Intermediate State of the Human KCNQ1 Channel Voltage-Sensor Domain

Biophysical Journal
Biophysics
2019English

Voltage Sensor Domain Mutations Involved in the Kv1.2 Channel Activation via MD Simulations

Biophysical Journal
Biophysics
2014English

A PQ-Channel Mutation Associated With Epilepsy Alters the Voltage Dependence of Channel Inactivation

Biophysical Journal
Biophysics
2015English

Gating Charge Interactions With the S1 Segment During Activation of a Na+ Channel Voltage Sensor

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2011English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy