Amanote Research
Register
Sign In
Atrogin-1 Increases Smooth Muscle Contractility Through Myocardin Degradation. Pavneet Singh, Dong Li, Yu Gui, Xi-Long Zheng
Journal of Cellular Physiology
- United States
doi 10.1002/jcp.25800
Full Text
Open PDF
Abstract
Available in
full text
Categories
Clinical Biochemistry
Cell Biology
Physiology
Date
July 25, 2017
Authors
Unknown
Publisher
Wiley
Related search
Atorvastatin Inhibits Myocardin Expression in Vascular Smooth Muscle Cells
Hypertension
Internal Medicine
Autonomic Modification of Intestinal Smooth Muscle Contractility
American Journal of Physiology - Advances in Physiology Education
Medicine
Physiology
Myocardin Is a Master Regulator of Smooth Muscle Gene Expression
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Hormone, Cytokine, and Nutritional Regulation of Sepsis-Induced Increases in Atrogin-1 and MuRF1 in Skeletal Muscle
American Journal of Physiology - Endocrinology and Metabolism
Endocrinology
Physiology
Metabolism
Diabetes
Atrogin-1 Affects Muscle Protein Synthesis and Degradation When Energy Metabolism Is Impaired by the Antidiabetes Drug Berberine
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Superoxide Anion Impairs Contractility in Cultured Aortic Smooth Muscle Cells
American Journal of Physiology - Heart and Circulatory Physiology
Cardiovascular Medicine
Physiology
Cardiology
The Serum Response Factor Coactivator Myocardin Is Required for Vascular Smooth Muscle Development
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Enhancement of Vascular Smooth Muscle Contractility by Alterations of Membranous Architecture
Anatomical Record
Evolution
Ecology
Systematics
Anatomy
Histology
Behavior
Biotechnology
Antagonistic Relationship Between the Unfolded Protein Response and Myocardin‐driven Transcription in Smooth Muscle
Journal of Cellular Physiology
Clinical Biochemistry
Cell Biology
Physiology