Nicotinamide Mononucleotide Attenuates Brain Injury After Intracerebral Hemorrhage by Activating Nrf2/Ho-1 Signaling Pathway

Scientific Reports- United Kingdom

doi 10.1038/s41598-017-00851-z

Full Text

Abstract

Available in

Categories

Multidisciplinary

Date

April 6, 2017

Authors

Publisher

Springer Science and Business Media LLC

Related search

Imatinib Inhibits Oxidative Stress Response in Spinal Cord Injury Rats by Activating Nrf2/Ho‑1 Signaling Pathway

Experimental and Therapeutic Medicine

Cancer Research

English

Inhibition of Activator Protein 1 Attenuates Neuroinflammation and Brain Injury After Experimental Intracerebral Hemorrhage

CNS Neuroscience and Therapeutics

English

Hyperbaric Oxygen Preconditioning Attenuates Brain Injury After Intracerebral Hemorrhage by Regulating Microglia Polarization in Rats

CNS Neuroscience and Therapeutics

English

Ischemic Preconditioning Attenuates Brain Edema After Experimental Intracerebral Hemorrhage

Translational Stroke Research

Cardiovascular Medicine

English

Toll-Like Receptor 4 Antagonist Attenuates Intracerebral Hemorrhage–Induced Brain Injury

Cardiovascular Medicine

Specialized Nursing

English

Targeting the Nrf2-Heme Oxygenase-1 Axis After Intracerebral Hemorrhage

Current Pharmaceutical Design

English

Ischemic Brain Injury After Intracerebral Hemorrhage: A Critical Review

Cardiovascular Medicine

Specialized Nursing

English

Gastrodin Protects Against LPS-induced Acute Lung Injury by Activating Nrf2 Signaling Pathway

English

Systemic Hemin Therapy Attenuates Blood–brain Barrier Disruption After Intracerebral Hemorrhage

Neurobiology of Disease

English