SM16, an Orally Active TGF- Type I Receptor Inhibitor Prevents Myofibroblast Induction and Vascular Fibrosis in the Rat Carotid Injury Model

Arteriosclerosis, Thrombosis, and Vascular Biology- United States

doi 10.1161/atvbaha.107.158030

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Date

April 1, 2008

Authors

P. Ann Boriack-Sjodin

Publisher

Ovid Technologies (Wolters Kluwer Health)

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SM16, an Orally Active TGF- Type I Receptor Inhibitor Prevents Myofibroblast Induction and Vascular Fibrosis in the Rat Carotid Injury Model

Arteriosclerosis, Thrombosis, and Vascular Biology- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

An Orally Active Chemokine Receptor CCR2 Antagonist Prevents Glomerulosclerosis and Renal Failure in Type 2 Diabetes

A Novel Small-Molecule Inhibitor of Transforming Growth Factor Type I Receptor Kinase (SM16) Inhibits Murine Mesothelioma Tumor Growth in Vivo and Prevents Tumor Recurrence After Surgical Resection

MEK Inhibition Prevents TGF‑β1‑induced Myofibroblast Transdifferentiation in Human Tenon Fibroblasts

Toll-Like Receptor 7-Mediated Type I Interferon Signaling Prevents Cholestasis- And Hepatotoxin-Induced Liver Fibrosis

In the Absence of Type III Receptor, the Transforming Growth Factor (TGF)-β Type II-B Receptor Requires the Type I Receptor to Bind TGF-β2

Orally Active Epoxyeicosatrienoic Acid Analog Attenuates Kidney Injury in Hypertensive Dahl Salt-Sensitive Rat

LCZ696 (Sacubitril/Valsartan), an Angiotensin-Receptor Neprilysin Inhibitor, Attenuates Cardiac Hypertrophy, Fibrosis and Vasculopathy in a Rat Model of Chronic Kidney Disease

Development of an ALK2-Biased BMP Type I Receptor Kinase Inhibitor

TACE-Mediated Ectodomain Shedding of the Type I TGF-β Receptor Downregulates TGF-β Signaling