Amanote Research
Register
Sign In
Cyclosporin A-Sensitive Induction of the Epstein-Barr Virus Lytic Switch Is Mediated via a Novel Pathway Involving a MEF2 Family Member
EMBO Journal
- Germany
doi 10.1093/emboj/16.1.143
Full Text
Open PDF
Abstract
Available in
full text
Categories
Immunology
Molecular Biology
Biochemistry
Microbiology
Neuroscience
Medicine
Genetics
Date
January 1, 1997
Authors
S. Liu
Publisher
Wiley
Related search
Initiation of Lytic DNA Replication in Epstein–Barr Virus: Search for a Common Family Mechanism
Future Virology
Virology
Cordycepin Is a Novel Chemical Suppressor of Epstein-Barr Virus Replication
Oncoscience
Cancer Research
Oncology
Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
A Cancer-Associated Epstein-Barr Virus BZLF1 Promoter Variant Enhances Lytic Infection
PLoS Pathogens
Immunology
Molecular Biology
Virology
Microbiology
Parasitology
Genetics
DNA-binding-defective Mutants of the Epstein-Barr Virus Lytic Switch Activator Zta Transactivate With Altered Specificities.
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Epstein-Barr Virus Lytic Replication Is Controlled by Posttranscriptional Negative Regulation of BZLF1.
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
The Epstein-Barr Virus Lytic Protein BZLF1 as a Candidate Target Antigen for Vaccine Development
Cancer immunology research
Cancer Research
Immunology
Unexpected Structure of Epstein–Barr Virus Lytic Cycle Activator Zta
Trends in Microbiology
Virology
Infectious Diseases
Microbiology
Epstein-Barr Virus Immediate-Early Protein BRLF1 Induces the Lytic Form of Viral Replication Through a Mechanism Involving Phosphatidylinositol-3 Kinase Activation
Journal of Virology
Insect Science
Immunology
Microbiology
Virology