(PDF) The 134.5 Protein of Herpes Simplex Virus 1 Complexes

The 134.5 Protein of Herpes Simplex Virus 1 Complexes With Protein Phosphatase 1 to Dephosphorylate the Subunit of the Eukaryotic Translation Initiation Factor 2 and Preclude the Shutoff of Protein Synthesis by Double-Stranded RNA-activated Protein Kinase

Proceedings of the National Academy of Sciences of the United States of America - United States

doi 10.1073/pnas.94.3.843

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Date

February 4, 1997

Authors

B. He

M. Gross

B. Roizman

Publisher

Proceedings of the National Academy of Sciences

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The 134.5 Protein of Herpes Simplex Virus 1 Complexes With Protein Phosphatase 1 to Dephosphorylate the Subunit of the Eukaryotic Translation Initiation Factor 2 and Preclude the Shutoff of Protein Synthesis by Double-Stranded RNA-activated Protein Kinase

Proceedings of the National Academy of Sciences of the United States of America - United States

doi 10.1073/pnas.94.3.843

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Regulation of Protein Synthesis: Activation by Double-Stranded RNA of a Protein Kinase That Phosphorylates Eukaryotic Initiation Factor 2

Second-Site Mutation Outside of the US10-12 Domain of 134.5 Herpes Simplex Virus 1 Recombinant Blocks the Shutoff of Protein Synthesis Induced by Activated Protein Kinase R and Partially Restores Neurovirulence

The Herpes Simplex Virus 1 Virion Host Shutoff Protein Enhances Translation of Viral Late mRNAs by Preventing mRNA Overload

Phosphorylation of the Cap-Binding Protein Eukaryotic Translation Initiation Factor 4E by Protein Kinase Mnk1 in Vivo

The 134.5 Protein of Herpes Simplex Virus 1 Is Required to Interfere With Dendritic Cell Maturation During Productive Infection

Herpes Simplex Virus Types 1 and 2 Induce Shutoff of Host Protein Synthesis by Different Mechanisms in Friend Erythroleukemia Cells.

The Herpes Simplex Virus Virion Host Shutoff Protein Enhances Translation of Viral True Late mRNAs Independently of Suppressing Protein Kinase R and Stress Granule Formation

RNA CUG-binding Protein 1 Increases Translation of 20-kDa Isoform of CCAAT/Enhancer-binding Protein Β by Interacting With the Α and Β Subunits of Eukaryotic Initiation Translation Factor 2

Inhibition of Human Immunodeficiency Virus Replication by the Herpes Simplex Virus Virion Host Shutoff Protein.