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Publications by A. Chariot
The Repressing Function of the Oncoprotein BCL-3 Requires CtBP, While Its Polyubiquitination and Degradation Involve the E3 Ligase TBLR1
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Induction of Nuclear Factor-κB and Its Downstream Genes by TNF-α and IL-1β Has a Pro-Apoptotic Role in Pancreatic Beta Cells
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Related publications
The IRAK-catalysed Activation of the E3 Ligase Function of Pellino Isoforms Induces the Lys63-Linked Polyubiquitination of IRAK1
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
The E3 Ligase TTC3 Facilitates Ubiquitination and Degradation of Phosphorylated Akt
Developmental Cell
Biochemistry
Developmental Biology
Genetics
Cell Biology
Molecular Biology
Structural Basis of the Activation and Degradation Mechanisms of the E3 Ubiquitin Ligase Nedd4L
Structure
Structural Biology
Molecular Biology
The E3 Ligase CHIP: Insights Into Its Structure and Regulation
BioMed Research International
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
PPARγ Is an E3 Ligase That Induces the Degradation of NFκB/p65
Nature Communications
Astronomy
Genetics
Molecular Biology
Biochemistry
Chemistry
Physics
Cholesterol Increases Protein Levels of the E3 Ligase MARCH6, and Thereby Stimulates Protein Degradation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Caveolin-3 Undergoes SUMOylation by the SUMO E3 Ligase PIASy
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Ubiquitination and Degradation of CFTR by the E3 Ubiquitin Ligase MARCH2 Through Its Association With Adaptor Proteins CAL and STX6
PLoS ONE
Multidisciplinary
The E3 Ubiquitin Ligase TRIM25 Regulates Adipocyte Differentiation via Proteasome-Mediated Degradation of PPARγ
Experimental and Molecular Medicine
Biochemistry
Medicine
Clinical Biochemistry
Molecular Biology
Molecular Medicine