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Publications by A. Kathrin Lutz
Loss of Parkin or PINK1 Function Increases Drp1-Dependent Mitochondrial Fragmentation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Reactive Oxygen Species Trigger Parkin/Pink1 Pathway-Dependent Mitophagy by Inducing Mitochondrial Recruitment of Parkin
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Inhibition of Drp1-Dependent Mitochondrial Fragmentation and Apoptosis by a Polypeptide Antagonist of Calcineurin
Cell Death and Differentiation
Cell Biology
Molecular Biology
Cadmium Induced Drp1-Dependent Mitochondrial Fragmentation by Disturbing Calcium Homeostasis in Its Hepatotoxicity
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Drosophila Parkin Requires PINK1 for Mitochondrial Translocation and Ubiquitinates Mitofusin
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Temporal Integration of Mitochondrial Stress Signals by the PINK1:Parkin Pathway
BMC Molecular and Cell Biology
Cell Biology
Molecular Biology
Loss of Parkin Impairs Mitochondrial Function and Leads to Muscle Atrophy
American Journal of Physiology - Cell Physiology
Physiology
Cell Biology
New Perception of Mitochondrial Regulatory Pathway in Parkinsonism €“ Ubiquitin, PINK1, and Parkin
Frontiers in Neurology
Neurology
A Novel USP30 Inhibitor Recapitulates Genetic Loss of USP30 and Sets the Trigger for PINK1-PARKIN Amplification of Mitochondrial Ubiquitylation.
Parkinsonism Due to Mutations in PINK1, Parkin, and DJ-1 and Oxidative Stress and Mitochondrial Pathways
Cold Spring Harbor perspectives in medicine
Biochemistry
Medicine
Genetics
Molecular Biology