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Publications by A. S. Corbin

KIT Signaling Governs Differential Sensitivity of Mature and Primitive CML Progenitors to Tyrosine Kinase Inhibitors

Cancer Research

Oncology

2013

English

Kinase Domain Mutants of BCR-Abl Exhibit Altered Transformation Potency, Kinase Activity, and Substrate Utilization, Irrespective of Sensitivity to Imatinib

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2006

English

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A Receptor Tyrosine Kinase cDNA Isolated From a Population of Enriched Primitive Hematopoietic Cells and Exhibiting Close Genetic Linkage to C-Kit.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1991

English

Point Mutation in Kit Receptor Tyrosine Kinase Reveals Essential Roles for Kit Signaling in Spermatogenesis and Oogenesis Without Affecting Other Kit Responses

English

Complementary Activity of Tyrosine Kinase Inhibitors Against Secondary Kit Mutations in Imatinib-Resistant Gastrointestinal Stromal Tumours

British Journal of Cancer

Cancer Research

Oncology

2019

English

MerReceptor Tyrosine Kinase Signaling

Journal of Biological Chemistry

English

Personalized Therapy of Tyrosine Kinase Inhibitors

Japanese Journal of Clinical Pharmacology and Therapeutics

Pharmacology

2013

English

Tyrosine Kinase Inhibitors for Diabetes

Science-Business eXchange

2008

English

Compound EGFR Mutations and Response to EGFR Tyrosine Kinase Inhibitors

Journal of Thoracic Oncology

English

Correction: Complementary Activity of Tyrosine Kinase Inhibitors Against Secondary Kit Mutations in Imatinib-Resistant Gastrointestinal Stromal Tumours

British Journal of Cancer

Cancer Research

Oncology

2019

English

EVI-1 Oncogene Expression Predicts Survival in Chronic-Phase CML Patients Resistant to Imatinib Treated With Second-Generation Tyrosine Kinase Inhibitors

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by A. S. Corbin

KIT Signaling Governs Differential Sensitivity of Mature and Primitive CML Progenitors to Tyrosine Kinase Inhibitors

Kinase Domain Mutants of BCR-Abl Exhibit Altered Transformation Potency, Kinase Activity, and Substrate Utilization, Irrespective of Sensitivity to Imatinib

Related publications

A Receptor Tyrosine Kinase cDNA Isolated From a Population of Enriched Primitive Hematopoietic Cells and Exhibiting Close Genetic Linkage to C-Kit.

Point Mutation in Kit Receptor Tyrosine Kinase Reveals Essential Roles for Kit Signaling in Spermatogenesis and Oogenesis Without Affecting Other Kit Responses

Complementary Activity of Tyrosine Kinase Inhibitors Against Secondary Kit Mutations in Imatinib-Resistant Gastrointestinal Stromal Tumours

MerReceptor Tyrosine Kinase Signaling

Personalized Therapy of Tyrosine Kinase Inhibitors

Tyrosine Kinase Inhibitors for Diabetes

Compound EGFR Mutations and Response to EGFR Tyrosine Kinase Inhibitors

Correction: Complementary Activity of Tyrosine Kinase Inhibitors Against Secondary Kit Mutations in Imatinib-Resistant Gastrointestinal Stromal Tumours

EVI-1 Oncogene Expression Predicts Survival in Chronic-Phase CML Patients Resistant to Imatinib Treated With Second-Generation Tyrosine Kinase Inhibitors