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Publications by Alan Kwok Shing Chiang

Role of ATM in the Formation of the Replication Compartment During Lytic Replication of Epstein-Barr Virus in Nasopharyngeal Epithelial Cells

Journal of Virology

English

Inhibition of Class I Histone Deacetylases by Romidepsin Potently Induces Epstein-Barr Virus Lytic Cycle and Mediates Enhanced Cell Death With Ganciclovir

International Journal of Cancer

Cancer Research

Oncology

2015

English

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Essential Role of Rta in Lytic DNA Replication of Epstein-Barr Virus

English

Latency and Lytic Replication in Epstein–Barr Virus-Associated Oncogenesis

Nature Reviews Microbiology

English

Epstein-Barr Virus Replication Within Pulmonary Epithelial Cells in Cryptogenic Fibrosing Alveolitis.

English

Epstein-Barr Virus Lytic Replication Is Controlled by Posttranscriptional Negative Regulation of BZLF1.

English

Initiation of Lytic DNA Replication in Epstein–Barr Virus: Search for a Common Family Mechanism

Future Virology

Virology

2010

English

Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication

English

Glycoprotein 110, the Epstein-Barr Virus Homolog of Herpes Simplex Virus Glycoprotein B, Is Essential for Epstein-Barr Virus Replication in Vivo.

English

Cordycepin Is a Novel Chemical Suppressor of Epstein-Barr Virus Replication

English

Reactivation of Lytic Replication From B Cells Latently Infected With Epstein-Barr Virus Occurs With High S-Phase Cyclin-Dependent Kinase Activity While Inhibiting Cellular DNA Replication

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Alan Kwok Shing Chiang

Role of ATM in the Formation of the Replication Compartment During Lytic Replication of Epstein-Barr Virus in Nasopharyngeal Epithelial Cells

Inhibition of Class I Histone Deacetylases by Romidepsin Potently Induces Epstein-Barr Virus Lytic Cycle and Mediates Enhanced Cell Death With Ganciclovir

Related publications

Essential Role of Rta in Lytic DNA Replication of Epstein-Barr Virus

Latency and Lytic Replication in Epstein–Barr Virus-Associated Oncogenesis

Epstein-Barr Virus Replication Within Pulmonary Epithelial Cells in Cryptogenic Fibrosing Alveolitis.

Epstein-Barr Virus Lytic Replication Is Controlled by Posttranscriptional Negative Regulation of BZLF1.

Initiation of Lytic DNA Replication in Epstein–Barr Virus: Search for a Common Family Mechanism

Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication

Glycoprotein 110, the Epstein-Barr Virus Homolog of Herpes Simplex Virus Glycoprotein B, Is Essential for Epstein-Barr Virus Replication in Vivo.

Cordycepin Is a Novel Chemical Suppressor of Epstein-Barr Virus Replication

Reactivation of Lytic Replication From B Cells Latently Infected With Epstein-Barr Virus Occurs With High S-Phase Cyclin-Dependent Kinase Activity While Inhibiting Cellular DNA Replication