Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by Anay Lázaro-Alfaro
Nanoencapsulated Quercetin Improves Cardioprotection During Hypoxia-Reoxygenation Injury Through Preservation of Mitochondrial Function
Oxidative Medicine and Cellular Longevity
Biochemistry
Medicine
Cell Biology
Aging
Related publications
Irisin Ameliorates Hypoxia/Reoxygenation-Induced Injury Through Modulation of Histone Deacetylase 4
PLoS ONE
Multidisciplinary
Sappanone a Alleviates Hypoxia/Reoxygenation-Induced Cardiomyocytes Injury Through Inhibition of Mitochondrial Apoptosis and Activation of PI3K-Akt-Gsk-3β Pathway.
Bioscience Reports
Biochemistry
Cell Biology
Molecular Biology
Biophysics
Brief Reoxygenation Episodes During Chronic Hypoxia Enhance Posthypoxic Recovery of LV Function
Basic Research in Cardiology
Cardiovascular Medicine
Physiology
Cardiology
Mitochondrial Complex I, Aconitase, and Succinate Dehydrogenase During Hypoxia-Reoxygenation: Modulation of Enzyme Activities by MnSOD
American Journal of Physiology - Lung Cellular and Molecular Physiology
Pulmonary
Respiratory Medicine
Physiology
Cell Biology
Baicalin Attenuates Liver Hypoxia/Reoxygenation Injury by Inducing Autophagy
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
PO2cycling Protects Diaphragm Function During Reoxygenation via ROS, Akt, ERK and Mitochondrial Channels
American Journal of Physiology - Cell Physiology
Physiology
Cell Biology
Luteolin Improves Heart Preservation Through Inhibiting Hypoxia‑dependent L‑type Calcium Channels in Cardiomyocytes
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
Protective Effect of Gap Junction Uncouplers Given During Hypoxia Against Reoxygenation Injury in Isolated Rat Hearts
American Journal of Physiology - Heart and Circulatory Physiology
Cardiovascular Medicine
Physiology
Cardiology
Picroside II Inhibits Hypoxia/Reoxygenation-Induced Cardiomyocyte Apoptosis by Ameliorating Mitochondrial Function Through a Mechanism Involving a Decrease in Reactive Oxygen Species Production
International Journal of Molecular Medicine
Medicine
Genetics