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Publications by Anil K. Bansal

Recruitment of Nuclear Factor Y to the Inverted CCAAT Element (ICE) by C-Jun and E1A Stimulates Basal Transcription of the Bone Sialoprotein Gene in Osteosarcoma Cells

Journal of Biological Chemistry

English

Related publications

Adenovirus E1A Protein Activates Transcription of the E1A Gene Subsequent to Transcription Complex Formation.

English

P300 and ATF-2 Are Components of the DRF Complex, Which Regulates Retinoic Acid- And E1a-Mediated Transcription of the C-Jun Gene in F9 Cells

Genes and Development

Genetics

Developmental Biology

1998

English

Metastasis‑associated Gene MAPK15 Promotes the Migration and Invasion of Osteosarcoma Cells via the C‑Jun/MMPs Pathway

English

The Role of an Inverted CCAAT Element in Transcriptional Activation of the Human DNA Topoisomerase IIα Gene by Heat Shock

Journal of Biological Chemistry

English

Corrigendum: Suppression of Vascular Permeability and Inflammation by Targeting of the Transcription Factor C-Jun

Nature Biotechnology

Applied Microbiology

Biotechnology

Biomedical Engineering

Molecular Medicine

Bioengineering

2015

English

Suppression of E1a-Mediated Transformation by the P50E4F Transcription Factor

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1999

English

Regulation of the Mouse Histone H2A.X Gene Promoter by the Transcription Factor E2F and CCAAT Binding Protein

Journal of Biological Chemistry

English

Mutation of the C-Fos Gene Dyad Symmetry Element Inhibits Serum Inducibility of Transcription in Vivo and the Nuclear Regulatory Factor Binding in Vitro.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1987

English

The Hepatocyte Nuclear Factor 3β Stimulates the Transcription of the Human Insulin-Like Growth Factor I Gene in a Direct and Indirect Manner

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Anil K. Bansal

Recruitment of Nuclear Factor Y to the Inverted CCAAT Element (ICE) by C-Jun and E1A Stimulates Basal Transcription of the Bone Sialoprotein Gene in Osteosarcoma Cells

Related publications

Adenovirus E1A Protein Activates Transcription of the E1A Gene Subsequent to Transcription Complex Formation.

P300 and ATF-2 Are Components of the DRF Complex, Which Regulates Retinoic Acid- And E1a-Mediated Transcription of the C-Jun Gene in F9 Cells

Metastasis‑associated Gene MAPK15 Promotes the Migration and Invasion of Osteosarcoma Cells via the C‑Jun/MMPs Pathway

The Role of an Inverted CCAAT Element in Transcriptional Activation of the Human DNA Topoisomerase IIα Gene by Heat Shock

Corrigendum: Suppression of Vascular Permeability and Inflammation by Targeting of the Transcription Factor C-Jun

Suppression of E1a-Mediated Transformation by the P50E4F Transcription Factor

Regulation of the Mouse Histone H2A.X Gene Promoter by the Transcription Factor E2F and CCAAT Binding Protein

Mutation of the C-Fos Gene Dyad Symmetry Element Inhibits Serum Inducibility of Transcription in Vivo and the Nuclear Regulatory Factor Binding in Vitro.

The Hepatocyte Nuclear Factor 3β Stimulates the Transcription of the Human Insulin-Like Growth Factor I Gene in a Direct and Indirect Manner