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Publications by Anna T. Tillmann
Redox Regulation, Rather Than Stress-Induced Phosphorylation, of a Hog1 Mitogen-Activated Protein Kinase Modulates Its Nitrosative-Stress-Specific Outputs
mBio
Virology
Microbiology
Related publications
MAPKKK-independent Regulation of the Hog1 Stress-Activated Protein Kinase inCandida Albicans
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Evidence for Involvement of Mitogen-Activated Protein Kinase, Rather Than Stress-Activated Protein Kinase, in Potentiation of 1-Β-D-Arabinofuranosylcytosine-Induced Apoptosis by Interruption of Protein Kinase C Signaling
Molecular Pharmacology
Molecular Medicine
Pharmacology
Regulation of Stress-Responsive Mitogen-Activated Protein (MAP) Kinase Pathways by TAO2
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
MKK7 Is a Stress-Activated Mitogen-Activated Protein Kinase Kinase Functionally Related Tohemipterous
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
The Hog1 Stress-Activated Protein Kinase Targets Nucleoporins to Control mRNA Export Upon Stress.
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
The Hog1 Mitogen-Activated Protein Kinase Is Essential in the Oxidative Stress Response and Chlamydospore Formation in Candidaalbicans
Eukaryotic Cell
MaHog1, a Hog1-Type Mitogen-Activated Protein Kinase Gene, Contributes to Stress Tolerance and Virulence of the Entomopathogenic Fungus Metarhizium Acridum
Microbiology
Microbiology
Mitogen-Activated Protein Kinase Hog1 in the Entomopathogenic Fungus Beauveria Bassiana Regulates Environmental Stress Responses and Virulence to Insects
Applied and Environmental Microbiology
Applied Microbiology
Biotechnology
Ecology
Food Science
Stress-Induced Phosphorylation of STAT1 at Ser727 Requires P38 Mitogen-Activated Protein Kinase Whereas IFN-gamma Uses a Different Signaling Pathway
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
