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Publications by Annette Balle Sørensen

Analysis of Wild-Type and Mutant SL3-3 Murine Leukemia Virus Insertions in the C-Myc Promoter During Lymphomagenesis Reveals Target Site Hot Spots, Virus-Dependent Patterns, and Frequent Error-Prone Gap Repair

Journal of Virology

English

Related publications

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

English

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

English

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

English

Atomic Force Microscopy Investigation of Wild-Type Moloney Murine Leukemia Virus Particles and Virus Particles Lacking the Envelope Protein

Virology

2004

English

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

English

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

English

Chimeric Human Immunodeficiency Virus Type 1 Containing Murine Leukemia Virus Matrix Assembles in Murine Cells

English

Herpes Simplex Virus Type 1 Promoted the Possibility of Leukemia Caused by L6565 Murine Leukemia Virus

The Open Veterinary Science Journal

2008

English

Reversion of a Moloney Murine Leukemia Virus RNase H Mutant at a Second Site Restores Enzyme Function and Infectivity.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Annette Balle Sørensen

Analysis of Wild-Type and Mutant SL3-3 Murine Leukemia Virus Insertions in the C-Myc Promoter During Lymphomagenesis Reveals Target Site Hot Spots, Virus-Dependent Patterns, and Frequent Error-Prone Gap Repair

Related publications

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

Atomic Force Microscopy Investigation of Wild-Type Moloney Murine Leukemia Virus Particles and Virus Particles Lacking the Envelope Protein

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

Chimeric Human Immunodeficiency Virus Type 1 Containing Murine Leukemia Virus Matrix Assembles in Murine Cells

Herpes Simplex Virus Type 1 Promoted the Possibility of Leukemia Caused by L6565 Murine Leukemia Virus

Reversion of a Moloney Murine Leukemia Virus RNase H Mutant at a Second Site Restores Enzyme Function and Infectivity.