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Publications by B. H. Shah
Gonadotrophin-Releasing Hormone Receptor Agonist-Mediated Down-Regulation of Gq Alpha/G11 Alpha (Pertussis Toxin-Insensitive) G Proteins in Alpha T3-1 Gonadotroph Cells Reflects Increased G Protein Turnover but Not Alterations in mRNA Levels.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Related publications
Close Association of the Alpha Subunits of Gq and G11 G Proteins With Actin Filaments in WRK1 Cells: Relation to G Protein-Mediated Phospholipase C Activation.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Orexin Receptor Type-1 Couples Exclusively to Pertussis Toxin-Insensitive G-Proteins, While Orexin Receptor Type-2 Couples to Both Pertussis Toxin-Sensitive and -Insensitive G-Proteins
Journal of Pharmacological Sciences
Molecular Medicine
Pharmacology
G Alpha 12 and G Alpha 13 Subunits Define a Fourth Class of G Protein Alpha Subunits.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Lipid Modifications of G Proteins: Alpha Subunits Are Palmitoylated.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Erratum mRNA and Protein Expression of Selective Alpha Subunits of G Proteins Are Abnormal in Prefrontal Cortex of Suicide Victims
Neuropsychopharmacology
Psychiatry
Mental Health
Pharmacology
Brain Α2-Adrenoceptors in Monoamine-Depleted Rats: Increased Receptor Density, G Coupling Proteins, Receptor Turnover and Receptor mRNA
British Journal of Pharmacology
Pharmacology
Role of Phospholipid Metabolism and G Protein in the Action Induced by Clostridium Perfringens Alpha-Toxin
Journal of Glycomics & Lipidomics
Stimulation of Human Thyroid Growth via the Inhibitory Guanine Nucleotide Binding (G) Protein Gi: Constitutive Expression of the G-Protein Alpha Subunit Gi Alpha-1 in Autonomous Adenoma.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
GH Substitution Reverses the Growth Phenotype but Not the Defective Ossification in Thyroid Hormone Receptor Alpha 1-/-Beta-/- Mice
Journal of Endocrinology
Endocrinology
Metabolism
Diabetes