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Publications by C. H. Thompson

Evolutionary and Functional Divergence Between the Cystic Fibrosis Transmembrane Conductance Regulator and Related ATP-binding Cassette Transporters

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2008

English

Abnormalities in Exercising Skeletal Muscle in Congestive Heart Failure Can Be Explained in Terms of Decreased Mitochondrial ATP Synthesis, Reduced Metabolic Efficiency, and Increased Glycogenolysis.

Heart

Cardiovascular Medicine

Cardiology

1996

English

Related publications

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Journal of Biological Chemistry

English

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Science-Business eXchange

2013

English

Commentary Cystic Fibrosis Transmembrane Conductance Regulator

Journal of General Physiology

Physiology

1997

English

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Science-Business eXchange

2012

English

Cystic Fibrosis Transmembrane Conductance Regulator and Adenosine Triphosphate

Science

Multidisciplinary

Philosophy of Science

History

1997

English

Optimization of the Degenerated Interfacial ATP Binding Site Improves the Function of Disease-Related Mutant Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Channels

Journal of Biological Chemistry

English

The Two ATP Binding Sites of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Play Distinct Roles in Gating Kinetics and Energetics

Journal of General Physiology

Physiology

2006

English

Low Beta-Adrenergic Sweat Responses in Cystic Fibrosis and Cystic Fibrosis Transmembrane Conductance Regulator-Related Metabolic Syndrome Children

Pediatric, Allergy, Immunology, and Pulmonology

English

Cystic Fibrosis Transmembrane Conductance Regulator Gene Mutation and Lung Cancer Risk

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by C. H. Thompson

Evolutionary and Functional Divergence Between the Cystic Fibrosis Transmembrane Conductance Regulator and Related ATP-binding Cassette Transporters

Abnormalities in Exercising Skeletal Muscle in Congestive Heart Failure Can Be Explained in Terms of Decreased Mitochondrial ATP Synthesis, Reduced Metabolic Efficiency, and Increased Glycogenolysis.

Related publications

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Commentary Cystic Fibrosis Transmembrane Conductance Regulator

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)

Cystic Fibrosis Transmembrane Conductance Regulator and Adenosine Triphosphate

Optimization of the Degenerated Interfacial ATP Binding Site Improves the Function of Disease-Related Mutant Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Channels

The Two ATP Binding Sites of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Play Distinct Roles in Gating Kinetics and Energetics

Low Beta-Adrenergic Sweat Responses in Cystic Fibrosis and Cystic Fibrosis Transmembrane Conductance Regulator-Related Metabolic Syndrome Children

Cystic Fibrosis Transmembrane Conductance Regulator Gene Mutation and Lung Cancer Risk