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Publications by C. Voytek

Spontaneous Reactivation of Herpes Simplex Virus Type 1 in Latently Infected Murine Sensory Ganglia

Journal of Virology

English

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Expression of the Herpes Simplex Virus Type 2 Latency-Associated Transcript Enhances Spontaneous Reactivation of Genital Herpes in Latently Infected Guinea Pigs

Journal of Experimental Medicine

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Antagonizing the Glucocorticoid Receptor Impairs Explant-Induced Reactivation in Mice Latently Infected With Herpes Simplex Virus 1

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Herpes Simplex Virus Type 1 Latently Infected Neurons Differentially Express Latency-Associated and ICP0 Transcripts

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The Role of LAT in Increased CD8+ T Cell Exhaustion in Trigeminal Ganglia of Mice Latently Infected With Herpes Simplex Virus 1

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An Avirulent ICP34.5 Deletion Mutant of Herpes Simplex Virus Type 1 Is Capable of in Vivo Spontaneous Reactivation.

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Monoclonal Antibodies Suppress Replication of Herpes Simplex Virus Type 1 in Trigeminal Ganglia.

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A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant With Increased Virulence and Reduced Spontaneous Reactivation

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The Herpes Simplex Virus 1 Latency-Associated Transcript Promotes Functional Exhaustion of Virus-Specific CD8+ T Cells in Latently Infected Trigeminal Ganglia: A Novel Immune Evasion Mechanism

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A 437-Base-Pair Deletion at the Beginning of the Latency-Associated Transcript Promoter Significantly Reduced Adrenergically Induced Herpes Simplex Virus Type 1 Ocular Reactivation in Latently Infected Rabbits.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by C. Voytek

Spontaneous Reactivation of Herpes Simplex Virus Type 1 in Latently Infected Murine Sensory Ganglia

Related publications

Expression of the Herpes Simplex Virus Type 2 Latency-Associated Transcript Enhances Spontaneous Reactivation of Genital Herpes in Latently Infected Guinea Pigs

Antagonizing the Glucocorticoid Receptor Impairs Explant-Induced Reactivation in Mice Latently Infected With Herpes Simplex Virus 1

Herpes Simplex Virus Type 1 Latently Infected Neurons Differentially Express Latency-Associated and ICP0 Transcripts

The Role of LAT in Increased CD8+ T Cell Exhaustion in Trigeminal Ganglia of Mice Latently Infected With Herpes Simplex Virus 1

An Avirulent ICP34.5 Deletion Mutant of Herpes Simplex Virus Type 1 Is Capable of in Vivo Spontaneous Reactivation.

Monoclonal Antibodies Suppress Replication of Herpes Simplex Virus Type 1 in Trigeminal Ganglia.

A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant With Increased Virulence and Reduced Spontaneous Reactivation

The Herpes Simplex Virus 1 Latency-Associated Transcript Promotes Functional Exhaustion of Virus-Specific CD8+ T Cells in Latently Infected Trigeminal Ganglia: A Novel Immune Evasion Mechanism

A 437-Base-Pair Deletion at the Beginning of the Latency-Associated Transcript Promoter Significantly Reduced Adrenergically Induced Herpes Simplex Virus Type 1 Ocular Reactivation in Latently Infected Rabbits.