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Publications by Caihui Cha

Endophilin 1 Knockdown Prevents Synaptic Dysfunction Induced by Oligomeric Amyloid Β

Molecular Medicine Reports

English

A Nuclear Localization Signal Is Required for the Nuclear Translocation of Fign and Its Microtubule‑severing Function

Molecular Medicine Reports

English

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Alzheimer's Disease Risk Factor Pyk2 Mediates Amyloid-Β Induced Synaptic Dysfunction and Loss

Journal of Neuroscience

Neuroscience

2018

English

A Prochelator Activated by Hydrogen Peroxide Prevents Metal-Induced Amyloid Β Aggregation

English

Non-Fibrillar Oligomeric Amyloid-Β Within Synapses

Journal of Alzheimer's Disease

English

Angiotensin-(1-7) Prevents Diabetes-Induced Cardiovascular Dysfunction

American Journal of Physiology - Heart and Circulatory Physiology

Cardiovascular Medicine

Physiology

Cardiology

2007

English

Protection Against Β-Amyloid-Induced Apoptosis by Peptides Interacting With Β-Amyloid

Journal of Biological Chemistry

English

Synaptic Plasticity Disruption by Amyloid Β Protein: Modulation by Potential Alzheimer's Disease Modifying Therapies: Figure 1

Biochemical Society Transactions

Biochemistry

2005

English

Selective Impairment of Some Forms of Synaptic Plasticity by Oligomeric Amyloid-Β Peptide in the Mouse Hippocampus: Implication of Extrasynaptic NMDA Receptors

Journal of Alzheimer's Disease

English

Neurotoxicity of Amyloid -Protein: Synaptic and Network Dysfunction

Cold Spring Harbor perspectives in medicine

English

Knockdown of Long Non Coding RNA SOX21-AS1 Attenuates Amyloid-Β-Induced Neuronal Damage by Sponging miR-107

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Caihui Cha

Endophilin 1 Knockdown Prevents Synaptic Dysfunction Induced by Oligomeric Amyloid Β

A Nuclear Localization Signal Is Required for the Nuclear Translocation of Fign and Its Microtubule‑severing Function

Related publications

Alzheimer's Disease Risk Factor Pyk2 Mediates Amyloid-Β Induced Synaptic Dysfunction and Loss

A Prochelator Activated by Hydrogen Peroxide Prevents Metal-Induced Amyloid Β Aggregation

Non-Fibrillar Oligomeric Amyloid-Β Within Synapses

Angiotensin-(1-7) Prevents Diabetes-Induced Cardiovascular Dysfunction

Protection Against Β-Amyloid-Induced Apoptosis by Peptides Interacting With Β-Amyloid

Synaptic Plasticity Disruption by Amyloid Β Protein: Modulation by Potential Alzheimer's Disease Modifying Therapies: Figure 1

Selective Impairment of Some Forms of Synaptic Plasticity by Oligomeric Amyloid-Β Peptide in the Mouse Hippocampus: Implication of Extrasynaptic NMDA Receptors

Neurotoxicity of Amyloid -Protein: Synaptic and Network Dysfunction

Knockdown of Long Non Coding RNA SOX21-AS1 Attenuates Amyloid-Β-Induced Neuronal Damage by Sponging miR-107