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Publications by Changlin Li
Protein Kinase D3 Is a Pivotal Activator of Pathological Cardiac Hypertrophy by Selectively Increasing the Expression of Hypertrophic Transcription Factors
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
A Radiomics Model to Predict the Invasiveness of Thymic Epithelial Tumors Based on Contrast‑enhanced Computed Tomography
Oncology Reports
Medicine
Cancer Research
Oncology
Related publications
The Transcription Factor GATA-6 Regulates Pathological Cardiac Hypertrophy
Circulation Research
Cardiovascular Medicine
Physiology
Cardiology
Differential Role of G Protein-Coupled Receptor Kinase 5 in Physiological Versus Pathological Cardiac Hypertrophy
Circulation Research
Cardiovascular Medicine
Physiology
Cardiology
A Ternary Complex of Transcription Factors, Nishéd and NFATc4, and Co-Activator P300 Bound to an Intronic Sequence, Intronic Regulatory Element, Is Pivotal for the Up-Regulation of Myosin Light Chain-2v Gene in Cardiac Hypertrophy
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
The TyrR Protein of Escherichia Coli Is a Class I Transcription Activator.
Journal of Bacteriology
Microbiology
Molecular Biology
Adult Cardiac Expression of the Activating Transcription Factor 3, ATF3, Promotes Ventricular Hypertrophy
PLoS ONE
Multidisciplinary
G Protein-Coupled Receptor Kinase 2 Promotes Cardiac Hypertrophy
PLoS ONE
Multidisciplinary
Reversal of Pathological Cardiac Hypertrophy via the MEF2-coregulator Interface
JCI insight
Medicine
Tripartite Motif 32 Prevents Pathological Cardiac Hypertrophy
Clinical Science
Medicine
Adrenergic Receptor Stimulation of the Mitogen-Activated Protein Kinase Cascade and Cardiac Hypertrophy
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
