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Publications by Chris J. Kershaw
ER Stress-Induced Reactive Oxygen Species (ROS) Are Detrimental for the Fitness of a Thioredoxin Reductase Mutant
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
DNA Damage-Induced Reactive Oxygen Species (ROS) Stress Response in Saccharomyces Cerevisiae
Free Radical Biology and Medicine
Biochemistry
Physiology
Mitochondrial Reactive Oxygen Species Are Obligatory Signals for Glucose-Induced Insulin Secretion
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Figure S2: Diverse Reactive Oxygen Species (ROS) Generation Levels
Editorial: Reactive Oxygen Species (ROS) Detection Methods in Biological System
Frontiers in Physiology
Physiology
NADPH-dependent Thioredoxin Reductase C Plays a Role in Nonhost Disease Resistance againstPseudomonas Syringaepathogens by Regulating Chloroplast-Generated Reactive Oxygen Species
PeerJ
Genetics
Molecular Biology
Biochemistry
Biological Sciences
Medicine
Agricultural
Neuroscience
Oxygen Matters: Hypoxia and Reactive Oxygen Species (ROS) as Pathophysiologic Mechanism in Rhinosinusitis
Nihon Bika Gakkai Kaishi (Japanese Journal of Rhinology)
An Endogenous Reactive Oxygen Species (ROS)-activated HDAC Inhibitor Prodrug for Cancer Chemotherapy
ChemMedChem
Organic Chemistry
Molecular Medicine
Pharmacology
Biochemistry
Toxicology
Drug Discovery
Pharmaceutics
Thioredoxin-2 Inhibits Mitochondrial Reactive Oxygen Species Generation and Apoptosis Stress Kinase-1 Activity to Maintain Cardiac Function
Circulation
Cardiovascular Medicine
Physiology
Cardiology
Activation of the AMPK-FOXO3 Pathway Reduces Fatty Acid-Induced Increase in Intracellular Reactive Oxygen Species by Upregulating Thioredoxin
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
